Results 121 to 130 of about 10,185,878 (314)
Molecular Oncology, EarlyView.This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi +6 more
wiley +1 more source
Faculty mentors for medical students
Academic Medicine, 1982 D H, Flach +3 more
openaire +3 more sources
Minutes: Medical School Faculty Advisory Council, March 5, 2002
, 2002 Faculty advisory council to the Dean of the Medical School.University of Minnesota. Medical School Faculty Advisory Council. (2002). Minutes: Medical School Faculty Advisory Council, March 5, 2002. Retrieved from the University Digital Conservancy, https:
University of Minnesota. Medical School Faculty Advisory Council
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ADP‐ribosylation: An emerging regulator of the epigenome
Molecular Oncology, EarlyView.ADP‐ribosylation has emerged as a dynamic epigenetic signaling mechanism that modifies histones and chromatin‐associated proteins. Through coordinated PARylation and MARylation, it integrates with other histone modifications to regulate chromatin structure, transcription factor activity, and gene expression, influencing genome function and disease ...
Cristel V. Camacho +2 more
wiley +1 more source
Minutes: Medical School Faculty Advisory Council, Dec. 5, 2006
, 2006 Faculty advisory council to the Dean of the Medical School.University of Minnesota. Medical School Faculty Advisory Council. (2006). Minutes: Medical School Faculty Advisory Council, Dec. 5, 2006. Retrieved from the University Digital Conservancy, https:/
University of Minnesota. Medical School Faculty Advisory Council
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Molecular Oncology, EarlyView.We show that emergence of castration‐resistant prostate (CRPC) is associated with significant upregulation of cyclins that positively regulate cyclin‐dependent kinase 2 (CDK2) and concomitant downregulation of CDK4 cyclins. This renders CRPC cells dependent on the high activity of CDK2, and CDK2 inhibitors synergistically sensitize CRPC cells to both ...
Joyeeta Chatterjee +3 more
wiley +1 more source
Minutes: Medical School Faculty Advisory Council, May 1, 2007
, 2007 Faculty advisory council to the Dean of the Medical School.University of Minnesota. Medical School Faculty Advisory Council. (2007). Minutes: Medical School Faculty Advisory Council, May 1, 2007. Retrieved from the University Digital Conservancy, https://
University of Minnesota. Medical School Faculty Advisory Council
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Molecular Oncology, EarlyView.Partial inhibition of focal adhesion kinase (FAK) can paradoxically promote tumor growth, rather than simply producing a weaker antitumor effect than that observed with strong FAK suppression. In breast cancer and melanoma models, targeting p110δ PI3K, particularly in macrophages, counteracted these tumor‐promoting effects, highlighting the importance ...
Lydia Xenou +4 more
wiley +1 more source
Minutes: Medical School Faculty Advisory Council, Nov. 5, 2002
, 2002 Faculty advisory council to the Dean of the Medical School.University of Minnesota. Medical School Faculty Advisory Council. (2002). Minutes: Medical School Faculty Advisory Council, Nov. 5, 2002. Retrieved from the University Digital Conservancy, https:/
University of Minnesota. Medical School Faculty Advisory Council
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