Results 121 to 130 of about 17,021,407 (316)

Northwestern University survey of Woman's Medical School Alumnae: Class of 1900.

open access: yes, 1914
Northwestern University Surveys of Woman's Medical School of Chicago Alumnae for the class of 1900, which were collected in 1914. These surveys contain the alumna's name, place and date of birth, education, hospital appointments, membership of medical ...

core  

Epigenetic silencing of the liver‐specific lncRNA LUNAR promotes liver cancer progression via NOTCH activation

open access: yesMolecular Oncology, EarlyView.
LUNAR is a liver‐specific long noncoding RNA (lncRNA) that is highly expressed in normal liver but becomes epigenetically silenced in hepatocellular carcinoma through promoter hypermethylation. Loss of LUNAR is associated with NOTCH activation, epithelial–mesenchymal transition, and metastasis, whereas restoring LUNAR restrains metastatic progression ...
Se Ha Jang   +9 more
wiley   +1 more source

Northwestern University Woman's Medical School 333-339 South Lincoln Street Chicago, Ill. Circular of information for 1896-97.

open access: yes, 1896
An annual circular of information from Northwestern University Woman's Medical School, previously known as the Woman's Medical College of Chicago, which discusses the curriculum, and includes a catalogue of ...

core  

Translating whole‐genome doubling into precision medicine in cancer

open access: yesMolecular Oncology, EarlyView.
Whole‐genome doubling creates a WGD‐positive tumor state characterized by persistent chromosomal instability, karyotypic diversification, and cellular stress. These same biological pressures drive aggressive tumor evolution while exposing therapeutic vulnerabilities, providing a rationale for WGD‐informed precision medicine. Whole‐genome doubling (WGD)
Sejung Lee, Junghyeok Lim, Jinhyuk Bhin
wiley   +1 more source

Northwestern University survey of Woman's Medical School Alumnae: Class of 1897.

open access: yes, 1914
Northwestern University Surveys of Woman's Medical School of Chicago Alumnae for the class of 1897, which were collected in 1914. These surveys contain the alumna's name, place and date of birth, education, hospital appointments, membership of medical ...

core  

Pharmacological chromatin remodeling enhances response to estrogen therapy in ER+ breast cancer

open access: yesMolecular Oncology, EarlyView.
Estrogen therapy elicits clinical benefit in ~ 30% of patients with endocrine‐resistant estrogen receptor (ER)‐positive breast cancer. Based on findings that ER transcriptional activation underlies response to estrogen therapy, we tested the effects of epigenetic dysregulation via pharmacological inhibition of histone deacetylases (HDACi).
Anneka L. Johnson Thomas   +16 more
wiley   +1 more source

Northwestern University survey of Woman's Medical School Alumnae: Class of 1892.

open access: yes, 1914
Northwestern University Surveys of Woman's Medical School of Chicago Alumnae for the class of 1892, which were collected in 1914. These surveys contain the alumna's name, place and date of birth, education, hospital appointments, membership of medical ...

core  

Spatial biology in cancer epigenetics

open access: yesMolecular Oncology, EarlyView.
Spatial epigenomics combines molecular profiling with tissue architecture to reveal how gene regulation is organized within intact tissues. In cancer, these technologies uncover the mechanisms driving tumor heterogeneity and microenvironmental interactions, opening new opportunities for biomarker discovery and precision medicine.
Eva Crespo‐García, Manel Esteller
wiley   +1 more source

Northwestern University Survery of Woman's Medical School Alumnae: Class of 1883.

open access: yes, 1914
Northwestern University Surveys of Woman's Medical School of Chicago Alumnae for the class of 1883, which were collected in 1914. These surveys contain the alumna's name, place and date of birth, education, hospital appointments, membership of medical ...

core  

Partial FAK suppression promotes tumor growth, an effect reversed by macrophage p110δ PI3K inactivation

open access: yesMolecular Oncology, EarlyView.
Partial inhibition of focal adhesion kinase (FAK) can paradoxically promote tumor growth, rather than simply producing a weaker antitumor effect than that observed with strong FAK suppression. In breast cancer and melanoma models, targeting p110δ PI3K, particularly in macrophages, counteracted these tumor‐promoting effects, highlighting the importance ...
Lydia Xenou   +4 more
wiley   +1 more source

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