Results 41 to 50 of about 1,272 (141)

Peer Review #2 of "Distribution of Porphyromonas gingivalis fimA and mfa1 fimbrial genotypes in subgingival plaques (v0.1)"

, 2018

semanticscholar   +3 more sources

Peer Review #1 of "Distribution of Porphyromonas gingivalis fimA and mfa1 fimbrial genotypes in subgingival plaques (v0.1)"

, 2018
A. Sharma
semanticscholar   +3 more sources

Porphyromonas gingivalis Fimbriae Induce Osteoclastogenesis via Toll-like Receptors in RAW264 Cells. [PDF]

Int J Mol Sci, 2022
The effect of Mfa1 fimbriae of Porphyromonas gingivalis on the progression of bone resorption remains unclear, especially compared with another fimbriae, FimA. We investigated the effect of Mfa1 on osteoclastogenesis together with FimA.
Suzuki Y, Kikuchi T, Goto H, Takayanagi Y, Kawamura S, Sawada N, Naiki Y, Kondo H, Hayashi JI, Hasegawa Y, Mitani A.   +10 more
europepmc   +2 more sources

Phylogenetic diversity in fim and mfa gene clusters between Porphyromonas gingivalis and Porphyromonas gulae, as a potential cause of host specificity

Journal of Oral Microbiology, 2020
Background Periodontopathic bacteria Porphyromonas gingivalis in humans and Porphyromonas gulae in animals are phylogenetically close and commonly have FimA and Mfa1 fimbriae.
Kaori Fujiwara-Takahashi, Takayasu Watanabe, Masahiro Shimogishi, Masaki Shibasaki, Makoto Umeda, Yuichi Izumi, Ichiro Nakagawa   +6 more
doaj   +1 more source

Porphyromonas gingivalis fimbriae

Journal of Oral Microbiology, 2013
Marginal periodontitis is not a homogeneous disease but is rather influenced by an intricate set of host susceptibility differences as well as diversities in virulence among the harbored organisms. It is likely that clonal heterogeneity of subpopulations
Morten Enersen, Kazuhiko Nakano, Atsuo Amano   +2 more
doaj   +1 more source

Disruption of Immune Homeostasis in Human Dendritic Cells via Regulation of Autophagy and Apoptosis by Porphyromonas gingivalis

Frontiers in Immunology, 2019
As fundamental processes of immune homeostasis, autophagy, and apoptosis must be maintained to mitigate risk of chronic inflammation and autoimmune diseases.
Mohamed M. Meghil, Mohamed M. Meghil, Omnia K. Tawfik, Mahmoud Elashiry, Mahmoud Elashiry, Mythilypriya Rajendran, Roger M. Arce, David J. Fulton, Patricia V. Schoenlein, Christopher W. Cutler   +9 more
doaj   +1 more source

Porphyromonas gingivalis Provokes Exosome Secretion and Paracrine Immune Senescence in Bystander Dendritic Cells

Frontiers in Cellular and Infection Microbiology, 2021
Periodontitis is a disease of ageing or inflammaging, and is comorbid with other more severe age-related chronic diseases. With advanced age comes an increase in accumulation of senescent cells that release soluble and insoluble pro-inflammatory factors ...
Ranya Elsayed, Mahmoud Elashiry, Yutao Liu, Ahmed El-Awady, Mark Hamrick, Christopher W. Cutler   +5 more
doaj   +1 more source

Polymerization and expression of Mfa1.

, 2015
(A) Immunoblot analysis using antiserum against Mfa1 fimbriae. Whole-cell lysates were obtained from P. gingivalis JI-1 (lane 1), FMFA4 (lane 2), and FMFA4C (lane 3). mfa1-deficient SMF-fimA (lane 4) was used as negative control.
Yoshiaki Hasegawa (136300), Noriyuki Kitai (803412), Ryota Ikai (803410), Fuminobu Yoshimura (136313), Yasuo Yoshida (31380), Yukitaka Murakami (136309), Keiji Nagano (136297), Richard J. Lamont (224455), Masashi Izumigawa (803411)   +8 more
core   +1 more source

Table_1_Maturation of the Mfa1 Fimbriae in the Oral Pathogen Porphyromonas gingivalis.DOCX

, 2018
The Mfa1 fimbriae of the periodontal pathogen Porphyromonas gingivalis are involved in adhesion, including binding to synergistic species in oral biofilms.
Leng Wu (5190419), Carolyn R. Casella (407749), Yoshiaki Hasegawa (136300), Daniel P. Miller (690349), Jae Y. Lee (2730721), Richard J. Lamont (224455)   +5 more
core   +1 more source

Tobacco upregulates P. gingivalis fimbrial proteins which induce TLR2 hyposensitivity.

PLoS ONE, 2010
BackgroundTobacco smokers are more susceptible to periodontitis than non-smokers but exhibit reduced signs of clinical inflammation. The underlying mechanisms are unknown.
Juhi Bagaitkar, Donald R Demuth, Carlo Amorin Daep, Diane E Renaud, Deanne L Pierce, David A Scott   +5 more
doaj   +1 more source