Results 31 to 40 of about 950 (126)
Background Periodontopathic bacteria Porphyromonas gingivalis in humans and Porphyromonas gulae in animals are phylogenetically close and commonly have FimA and Mfa1 fimbriae.
Kaori Fujiwara-Takahashi +6 more
doaj +1 more source
The human fungal pathogen Candida albicans can switch stochastically and heritably between a “white” phase and an “opaque” phase. Opaque cells are the mating-competent form of the species, whereas white cells are thought to be essentially “sterile”. Here,
Guobo Guan +6 more
doaj +1 more source
Porphyromonas gingivalis fimbriae
Marginal periodontitis is not a homogeneous disease but is rather influenced by an intricate set of host susceptibility differences as well as diversities in virulence among the harbored organisms. It is likely that clonal heterogeneity of subpopulations
Morten Enersen +2 more
doaj +1 more source
Tobacco smoke augments Porphyromonas gingivalis-Streptococcus gordonii biofilm formation.
Smoking is responsible for the majority of periodontitis cases in the US and smokers are more susceptible than non-smokers to infection by the periodontal pathogen Porphyromonas gingivalis. P.
Juhi Bagaitkar +5 more
doaj +1 more source
Structural studies of the five pilin proteins building up the type-V pilus Mfa1 of Porphyromonas gingivalis [PDF]
Structural studies of the five pilin proteins building up the type-V pilus Mfa1 of Porphyromonas ...
Karina Persson +3 more
openaire +1 more source
Signaling via pattern recognition receptors (PRRs) expressed on professional antigen presenting cells, such as dendritic cells (DCs), is crucial to the fate of engulfed microbes.
Ahmed R El-Awady +11 more
doaj +1 more source
Periodontitis is a disease of ageing or inflammaging, and is comorbid with other more severe age-related chronic diseases. With advanced age comes an increase in accumulation of senescent cells that release soluble and insoluble pro-inflammatory factors ...
Ranya Elsayed +5 more
doaj +1 more source
As fundamental processes of immune homeostasis, autophagy, and apoptosis must be maintained to mitigate risk of chronic inflammation and autoimmune diseases.
Mohamed M. Meghil +9 more
doaj +1 more source
Tobacco upregulates P. gingivalis fimbrial proteins which induce TLR2 hyposensitivity.
BackgroundTobacco smokers are more susceptible to periodontitis than non-smokers but exhibit reduced signs of clinical inflammation. The underlying mechanisms are unknown.
Juhi Bagaitkar +5 more
doaj +1 more source
Oral community interactions of Filifactor alocis in vitro. [PDF]
Filifactor alocis is a gram positive anaerobe that is emerging as an important periodontal pathogen. In the oral cavity F. alocis colonizes polymicrobial biofilm communities; however, little is known regarding the nature of the interactions between F ...
Qian Wang +4 more
doaj +1 more source

