Results 201 to 210 of about 212,558 (382)

Bedside Assessment of Downgaze Limit by Amplitude

open access: yesMovement Disorders Clinical Practice, EarlyView.
Abstract Background Bedside assessment of downgaze palsy could be difficult, due to the lack of a marker to differentiate normal from palsy, which could delay the diagnosis of progressive supranuclear palsy (PSP). Amplitude based bedside assessment using the intercanthal line as a marker was reported to easily identify, quantify, and record downgaze ...
Tao Xie   +6 more
wiley   +1 more source

Midbrain-like Organoids from Human Pluripotent Stem Cells Contain Functional Dopaminergic and Neuromelanin-Producing Neurons.

open access: yesCell Stem Cell, 2016
J. Jo   +22 more
semanticscholar   +1 more source

Modeling G2019S-LRRK2 Sporadic Parkinson's Disease in 3D Midbrain Organoids

open access: yesStem Cell Reports, 2019
Hongwon Kim   +9 more
semanticscholar   +1 more source

Cholinergic Inhibition of Ventral Midbrain Dopamine Neurons [PDF]

open access: bronze, 2000
Christopher D. Fiorillo   +1 more
openalex   +1 more source

Brain Imaging Changes Following Deep Brain Stimulation Patients with Parkinson's Disease: A Literature Review

open access: yesMovement Disorders Clinical Practice, EarlyView.
Abstract Background Parkinson's Disease (PD) is a progressive neurodegenerative disorder primarily characterized by motor symptoms such as tremors, rigidity, and bradykinesia. Structural brain changes, including atrophy in the midbrain, basal ganglia, and cortical regions such as the frontal and temporal lobes, are observed in advanced stages.
Suraiya Mangra   +5 more
wiley   +1 more source

Dopamine measurement in midbrain organoids by HPLC v1 [PDF]

open access: gold
Hariam Raji   +1 more
openalex   +1 more source

Compound Heterozygous Structural Variants in Cases with Unsolved PRKN‐Associated Parkinson's Disease

open access: yesMovement Disorders, EarlyView.
Abstract Background Biallelic mutations in the PRKN gene are a common cause of early‐onset Parkinson's disease (EOPD). In addition to single nucleotide variants, structural variants contribute substantially to the mutational profile of PRKN. A significant portion of patients with EOPD remains genetically unsolved.
Agata Fant   +24 more
wiley   +1 more source

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