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The oxidase systems of Ascaris-muscle mitochondria

Biochimica et Biophysica Acta (BBA) - Bioenergetics, 1970
Abstract 1. The oxidase systems of Ascaris-muscle mitochondria were investigated by polarographic and spectroscopic techniques. 2. Spectrophotometric studies at −196° revealed that Ascaris-muscle mitochondria contained low levels of substrate-reducible a-, b- and c-type cytochromes.
K S, Cheah, B, Chance
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Giant Mitochondria in Human Muscle With Inclusions

Archives of Neurology, 1967
RECENTLY, two reports have been published on myopathies characterized primarily by abnormalities of mitochondria. In the first, Luft et al1described a severely hypermetabolic patient with loosely coupled oxidative phosphorylation in mitochondria and, in the second, Shy et al2described pleoconial and megaconial conditions, both related to aberrent ...
S A, Shafiq, A T, Milhorat, M A, Gorycki
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Oxidative phosphorylation in Moniezia muscle mitochondria

Biochimica et Biophysica Acta (BBA) - Bioenergetics, 1971
Abstract 1. 1. Mitochondria isolated from Moniezia expansa , an intestinal parasitic tapeworm, have the same structural characteristics as the classical mammalian aerobic mitochondria by having clearly defined outer and inner membranes, outer compartment and intracristal spaces, and elementary particles attached to the mitochondrial cristae. 2.
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Neuromuscular Disorders with Abnormal Muscle Mitochondria

1980
Publisher Summary Mitochondria are the site of oxidative phosphorylation of adenosine diphosphate (ADP) to adenosine triphosphate (ATP). The mechanical work of muscle fibers—that is, shortening of myofibrils, depends on an adequate supply of ATP, and metabolic disorders of mitochondria can be assumed to impair the function of the muscle fiber.
Z, Kamieniecka, H, Schmalbruch
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Mitochondria in Skeletal Muscle

Exercise and Sport Sciences Reviews, 2008
Apoptosis is an essential process that plays a critical role in both tissue development and maintenance. Apoptosis has been shown to be involved in skeletal muscle atrophy resulting from chronic muscular disuse, sarcopenia, and mitochondrial myopathies. Exercise may attenuate some of the proapoptotic adaptations that occur during these conditions. This
Peter J, Adhihetty   +2 more
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String mitochondria in mouse soleus muscle

Microscopy Research and Technique, 2012
Red myofibers in mouse soleus muscle have two spatially distinct populations of mitochondria: one where these organelles are disposed in large clusters just inside the sarcolemma and the other situated between the myofibrils. In most cases, the interfibrillar mitochondria (IFM), which are much smaller than the subsarcolemmal ones (SSM), are arranged as
Hisashi, Fujioka   +4 more
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Mitochondria in Muscle and Exercise

2017
This chapter will discuss the role of mitochondria in muscle, particularly the effect of acute and chronic exercise. The focus will be on human studies. A general overview of mitochondrial structure and function will be followed by a discussion of the diverse techniques used to measure mitochondrial function.
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Volume rendering of heart muscle mitochondria

Proceedings of the 2011 companion on High Performance Computing Networking, Storage and Analysis Companion, 2011
Volumetric representations of electron microscopy reveal nanometer structural features of the mitochondrion. These organelles are the cell's 'power plant,' generating most of the adenosine triphosphate - the main source of chemical energy for a cell. Visualized here are the mitochondria present in human heart muscle.
Brandt M. Westing   +2 more
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Respiratory control in cockroach-muscle mitochondria

Biochimica et Biophysica Acta, 1963
Abstract Mitochondria isolated from cockroach thoracic muscles have been used to demonstrate that the rate of oxidation of several Krebs-cycle intermediates is under the control of adenosine diphosphate concentration. Respiratory-control indices as high as 25 have been achieved with both pyruvate and glutamate.
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Skeletal Muscle Mitochondria: The Aerobic Gate?

1988
At an animal's maximum aerobic capacity (VO2max), the O2 flowing through the respiratory system is consumed by a functionally exclusive sink, skeletal muscle mitochondria. Thus, O2 consumption will never exceed the muscles O2 demand. If the system is ideally designed, structures upstream to the skeletal muscle O2 sink must be built to insure adequate ...
S L, Lindstedt, D J, Wells
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