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Mitochondria: Muscle Morphology
2019Mitochondrial genetic defects can potentially affect any cell, tissue, and organ; however, they are often referred to as mitochondrial encephalomyopathies to indicate that skeletal muscle and brain tissue, due to their high energy demand, are most often involved.
Monica Sciacco +4 more
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Familial Myopathy With Abnormal Muscle Mitochondria
Archives of Neurology, 1968DURING the past ten years several myopathies with unusual microscopic changes in muscle have been described. 1-3 Shy and Magee presented the original report of a familial myopathy, "central core disease," characterized by weakness and hypotonia from birth and a characteristic morphologic abnormality in the center of muscle fibers.
A N, D'Agostino +3 more
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Mitochondria in muscle cell death
Neurological Sciences, 1999Mitochondria, the main source of energy for eukaryotic cells through oxidative phosphorylation, also play a key role in the pathways to cell death. The mode of cell death may be influenced by the availability of ATP, and its very occurrence may critically depend on release of mitochondrial proteins like cytochrome c, apoptosis-inducing factor and ...
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String mitochondria in mouse soleus muscle
Microscopy Research and Technique, 2012Red myofibers in mouse soleus muscle have two spatially distinct populations of mitochondria: one where these organelles are disposed in large clusters just inside the sarcolemma and the other situated between the myofibrils. In most cases, the interfibrillar mitochondria (IFM), which are much smaller than the subsarcolemmal ones (SSM), are arranged as
Hisashi, Fujioka +4 more
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[14] Skeletal muscle mitochondria
1967Publisher Summary Bulk of the muscle tissue consists of myofibrils, and the mitochondria, as a rule, constitute a much smaller portion of the total mass of the tissue than in other organs. A crucial difference in the preparation of mitochondria from skeletal muscle as compared with other tissues is that sucrose, or in general, nonelectrolytes, cannot
Lars Ernster, Kerstin Nordenbrand
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Neuromuscular Disorders with Abnormal Muscle Mitochondria
1980Publisher Summary Mitochondria are the site of oxidative phosphorylation of adenosine diphosphate (ADP) to adenosine triphosphate (ATP). The mechanical work of muscle fibers—that is, shortening of myofibrils, depends on an adequate supply of ATP, and metabolic disorders of mitochondria can be assumed to impair the function of the muscle fiber.
Z, Kamieniecka, H, Schmalbruch
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Oxidative phosphorylation in Moniezia muscle mitochondria
Biochimica et Biophysica Acta (BBA) - Bioenergetics, 1971Abstract 1. 1. Mitochondria isolated from Moniezia expansa , an intestinal parasitic tapeworm, have the same structural characteristics as the classical mammalian aerobic mitochondria by having clearly defined outer and inner membranes, outer compartment and intracristal spaces, and elementary particles attached to the mitochondrial cristae. 2.
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Skeletal Muscle Mitochondria: The Aerobic Gate?
1988At an animal's maximum aerobic capacity (VO2max), the O2 flowing through the respiratory system is consumed by a functionally exclusive sink, skeletal muscle mitochondria. Thus, O2 consumption will never exceed the muscles O2 demand. If the system is ideally designed, structures upstream to the skeletal muscle O2 sink must be built to insure adequate ...
S L, Lindstedt, D J, Wells
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Mitochondria in Skeletal Muscle
Exercise and Sport Sciences Reviews, 2008Apoptosis is an essential process that plays a critical role in both tissue development and maintenance. Apoptosis has been shown to be involved in skeletal muscle atrophy resulting from chronic muscular disuse, sarcopenia, and mitochondrial myopathies. Exercise may attenuate some of the proapoptotic adaptations that occur during these conditions. This
Peter J, Adhihetty +2 more
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Respiratory control in cockroach-muscle mitochondria
Biochimica et Biophysica Acta, 1963Abstract Mitochondria isolated from cockroach thoracic muscles have been used to demonstrate that the rate of oxidation of several Krebs-cycle intermediates is under the control of adenosine diphosphate concentration. Respiratory-control indices as high as 25 have been achieved with both pyruvate and glutamate.
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