Results 61 to 70 of about 237,811 (262)

Hypertrophy Stimulation at the Onset of Type I Diabetes Maintains the Soleus but Not the EDL Muscle Mass in Wistar Rats

open access: yesFrontiers in Physiology, 2017
Diabetes mellitus induces a reduction in skeletal muscle mass and strength. Strength training is prescribed as part of treatment since it improves glycemic control and promotes increase of skeletal muscle mass. The mechanisms involved in overload-induced
Marco A. S. Fortes   +7 more
doaj   +1 more source

Sarcoplasmic Hypertrophy in Skeletal Muscle: A Scientific “Unicorn” or Resistance Training Adaptation?

open access: yesFrontiers in Physiology, 2020
Skeletal muscle fibers are multinucleated cells that contain mostly myofibrils suspended in an aqueous media termed the sarcoplasm. Select evidence suggests sarcoplasmic hypertrophy, or a disproportionate expansion of the sarcoplasm relative to myofibril
Michael D. Roberts   +6 more
doaj   +1 more source

Mouse skeletal muscle fiber-type-specific macroautophagy and muscle wasting are regulated by a Fyn/STAT3/Vps34 signaling pathway [PDF]

open access: yes, 2012
Skeletal muscle atrophy induced by aging (sarcopenia), inactivity, and prolonged fasting states (starvation) is predominantly restricted to glycolytic type II muscle fibers and typical spares oxidative type I fibers.
Alessi   +49 more
core   +2 more sources

Myonuclear Domain Flexibility Challenges Rigid Assumptions on Satellite Cell Contribution to Skeletal Muscle Fiber Hypertrophy

open access: yesFrontiers in Physiology, 2018
Satellite cell-mediated myonuclear accretion is thought to be required for skeletal muscle fiber hypertrophy, and even drive hypertrophy by preceding growth. Recent studies in humans and rodents provide evidence that challenge this axiom.
Kevin A. Murach   +9 more
doaj   +1 more source

Do the anatomical and physiological properties of a muscle determine its adaptive response to different loading protocols?

open access: yesPhysiological Reports, 2020
It has been proposed that superior muscle hypertrophy may be obtained by training muscles predominant in type I fibers with lighter loads and those predominant in type II fibers with heavier loads.
Brad J. Schoenfeld   +8 more
doaj   +1 more source

Autophagy signaling in hypertrophied muscles of diabetic and control rats

open access: yesFEBS Open Bio, 2023
Autophagy plays a vital role in cell homeostasis by eliminating nonfunctional components and promoting cell survival. Here, we examined the levels of autophagy signaling proteins after 7 days of overload hypertrophy in the extensor digitorum longus (EDL)
Maria V. M. Scervino   +10 more
doaj   +1 more source

The effect of growth hormone on the growth of the tibia/fibula complex and femurs of hypophysectomized rats after unilateral limb denervation [PDF]

open access: yes, 1972
Thesis (M.Sc.D.)--Boston University School of Graduate Dentistry, 1972 (Orthodontics)Bibliography ...
Kapit, Arthur L., Oliveira, Lawrence J.
core  

Beware the Meta-Analysis: Is Multiple Set Training Really Better than Single-Set Training for Muscle Hypertrophy? [PDF]

open access: yes, 2012
While many individuals perform resistance training with the purpose of increasing the size of their muscles, reviews have not clarified a training prescription for optimizing hypertrophy. A 2010 meta-analysis concluded that multiple set training resulted
Fisher, James
core   +1 more source

Micro-RNA family that modulates fibrosis and uses thereof [PDF]

open access: yes, 2017
The present invention relates to the identification of a microRNA family, designated miR-29a-c, that is a key regulator of fibrosis in cardiac tissue. The inventors show that members of the miR-29 family are down-regulated in the heart tissue in response
Olson, Eric N., van Rooij, Eva
core   +1 more source

Micro-RNAS that control myosin expression and myofiber identity [PDF]

open access: yes, 2015
The present invention relates to the identification of two microRNAs, miR-499 and miR-208b, that repress fast skeletal muscle contractile protein genes. Expression of miR-499 and/or miR-208b can be used to repress fast fiber genes and activate slow fiber
Olson, Eric N., van Rooij, Eva
core   +1 more source

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