Results 221 to 230 of about 50,819 (268)

Limited adoption of thymectomy for nonthymomatous myasthenia gravis. [PDF]

open access: yesJ Thorac Dis
Carrera D   +4 more
europepmc   +1 more source

Optimizing myasthenia gravis management in practice: integrating clinical scales and classification systems. [PDF]

open access: yesArq Neuropsiquiatr
Baima JPS   +6 more
europepmc   +1 more source

Effect of CXCR5, PD-1 and ICOS on B-cell responses and relevance to myasthenia gravis. [PDF]

open access: yesClin Transl Immunology
Çebi M   +4 more
europepmc   +1 more source

Myasthenia Gravis [PDF]

open access: yesSeminars in Neurology, 2015
Myasthenia gravis is an autoimmune neuromuscular disease caused by antibodies directed against the postsynaptic muscle membrane. The clinical hallmark of the disease is fluctuating and fatigable weakness that affects the ocular muscles (resulting in diplopia and ptosis), the bulbar muscles (causing dysphagia, dysarthria, and dyspnea), and extremity ...
Kelly G, Gwathmey, Ted M, Burns
core   +4 more sources

Myasthenia gravis

open access: yesJAAPA
Myasthenia gravis (MG) is a rare neuromuscular junction disorder that is characterized by fatigable weakness of muscles. People with MG experience various clinical manifestations based on the muscles involved. MG can be autoimmune, paraneoplastic, congenital, medication-related, or transient in the neonatal period due to the passive placental transfer ...
Mamatha, Pasnoor   +2 more
core   +5 more sources
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Myasthenia Gravis

Seminars in Neurology, 1990
The treatment of patients with myasthenia gravis should be individualized according to the extent (ocular versus generalized) and severity (mild to severe) of disease, the presence or absence of concomitant disease (including but not limited to other autoimmune diseases and thymoma), and, to a lesser degree, the age of the patient. Thymectomy should be
D M, Linton, D, Philcox
openaire   +4 more sources

Myasthenia Gravis

New England Journal of Medicine, 2016
Myasthenia gravis is an autoimmune disease, which leads to load-dependent weakness of voluntary skeletal muscles with recovery of function after resting. The disease is caused by autoantibodies directed against the postsynaptic nicotinic acetylcholine receptors (AChR) leading to a reduction of neuromuscular transmission.
Sabina, Miranda   +2 more
openaire   +8 more sources

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