Results 291 to 300 of about 108,757 (334)

Axon Myelination: Myelination without myelin-associated glycoprotein

Current Biology, 1994
Mice lacking myelin-associated glycoprotein surprisingly myelinate almost normally but their oligodendrocytes have lost their periaxonal cytoplasmic 'collars' and accidentally myelinate already-myelinated axons.
A, Meyer-Franke, B, Barres
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Myelin, myelin-related disorders, and psychosis

Schizophrenia Research, 2015
The neuropathological basis of schizophrenia and related psychoses remains elusive despite intensive scientific investigation. Symptoms of psychosis have been reported in a number of conditions where normal myelin development is interrupted. The nature, location, and timing of white matter pathology seem to be key factors in the development of ...
Michelle I, Mighdoll, Ran, Tao, Joel E, Kleinman, Thomas M, Hyde   +3 more
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Myelination and Retarded Myelination

1989
Flechsig (1920) was the originator of the view that the degree of myelination of the central nervous system might be correlated with functional capacity. In his theory he stated that myelination started in projection pathways before association pathways, in peripheral nerves before central pathways, and in sensory areas before motor ones.
Jacob Valk, Marjo S. van der Knaap
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Myelinated Nerve Fibre, Myelin

2010
Nerve fibres designed for particularly rapid and efficient conduction of action potentials are equipped with a myelin sheath, a lipid-enriched layer, produced by specialised glial cells, the oligodendrocytes in the central nervous system, and the Schwann cells in the peripheral nervous system.
Margit Pavelka, Jürgen Roth
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Myelin repair: developmental myelination redux?

Nature Neuroscience, 2005
A recent study of an Olig1 knockout mouse concludes that remyelination after injury may occur by a different mechanism from myelination during normal development, but another report suggests that this mouse model should be interpreted cautiously.
Roumen Balabanov, Brian Popko
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Myelin‐Deficient Rat: Analysis of Myelin Proteins

Journal of Neurochemistry, 1986
Abstract: Myelin basic protein (BP), proteolipid protein (PLP), myelin‐associated glycoprotein (MAG), and 2′,3′‐cyclic nucleotide 3′‐phosphodiesterase (CNPase) activity were quantitated in the brains and spinal cords of normal and myelin‐deficient (md) rats at 8, 12, 18, and 25 days of age.
K, Yanagisawa, I D, Duncan, J P, Hammang, R H, Quarles   +3 more
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