Results 211 to 220 of about 68,817 (283)

Molecular Alterations in Ferroptosis and the Effects of Resveratrol: A Systematic Review

open access: yesJournal of Biochemical and Molecular Toxicology, Volume 39, Issue 7, July 2025.
Ferroptosis is a cell death mechanism characterized by iron overload, accumulation of reactive oxygen species, deficits in glutathione metabolism, lipid peroxidation, and mitochondrial impairments. Resveratrol can prevent or reverse these alterations by chelating iron and upregulating antioxidant defenses. ABSTRACT Ferroptosis is an iron‐dependent cell
Ana Beatriz Dos Santos   +4 more
wiley   +1 more source

Loss of Intracellular Dystrophin-A Potential Mechanism for Myocardial Reperfusion Injury-

open access: bronze, 2003
Shiori Kyoi   +8 more
openalex   +2 more sources

The Interplay of Cross‐Organ Immune Regulation in Inflammation and Cancer

open access: yesMedComm, Volume 6, Issue 7, July 2025.
Organs interact dynamically through immune regulation, influencing systemic immune responses and disease progression. We review molecular and cellular mechanisms of cross‐organ immune regulation in inflammation and cancer, focusing on gut, liver, and brain interactions with other organs.
Jie Dou   +6 more
wiley   +1 more source

MicroRNA‐100‐5p Exacerbates Myocardial Ischemia–Reperfusion Injury Through Downregulation of PRMT5 [PDF]

open access: yesFASEB J
MiR‐100‐5p overexpression downregulates PRMT5, leading to increased PTEN activity and reduced AKT signaling. This molecular cascade promotes cardiomyocyte apoptosis, enhances fibrotic remodeling, and exacerbates cardiac dysfunction following myocardial ischemia–reperfusion injury.
Wang Z   +9 more
europepmc   +2 more sources

Macrophage Signaling Pathways in Health and Disease: From Bench to Bedside Applications

open access: yesMedComm, Volume 6, Issue 7, July 2025.
Macrophage functions in phagocytosis, tissue repair, immune regulation, and neuronal network protection. And graphic abract image in the attachment. ABSTRACT Macrophages exhibit remarkable functional plasticity by dynamically polarizing into proinflammatory or antiinflammatory subsets in response to microenvironmental cues. This duality underpins their
Yongquan Chi   +6 more
wiley   +1 more source

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