Results 21 to 30 of about 14,187 (322)

Non-invasive intradialytic percutaneous perfusion monitoring: a view to the heart through the skin

open access: yesFrontiers in Nephrology, 2023
IntroductionThe life-sustaining treatment of hemodialysis (HD) induces recurrent and cumulative systemic circulatory stress resulting in cardiovascular injury.
Jarrin D. Penny   +12 more
doaj   +1 more source

Myocardial stunning in hyperthyroidism [PDF]

open access: yesClinical Cardiology, 2000
AbstractThe cases of two patients with hyperthyroidism and acute left ventricular (LV) dysfunction with segmental wall motion abnormalities resulting in heart failure are reported. Both had electrocardiographic changes mimicking ischemic coronary artery disease.
N, Pereira   +5 more
openaire   +2 more sources

Toxic Myocardial Injury in a Patient With Coronary Atherosclerosis, Caused by Acute Poisoning With Gaseous Chlorine

open access: yesНеотложная медицинская помощь, 2021
Acute chlorine gas poisoning leads to activation of the sympathetic nervous system and, as a consequence, dysfunction of the cardiovascular system (CVS). We report a clinical case of toxic myocardial injury with gaseous  chlorine.
K. G. Alidzhanova   +5 more
doaj   +1 more source

ACE-inhibition prevents postischemic coronary leukocyte adhesion and leukocyte-dependent reperfusion injury [PDF]

open access: yes, 1997
Objective: Polymorphonuclear leukocytes (PMN), retained in the microvascular bed, can contribute to postischemic myocardial reperfusion injury. Since a beneficial effect of ACE-inhibition on reperfusion injury has been reported, we investigated the ...
Becker, Bernhard F.   +6 more
core   +1 more source

Myocardial stunning after resuscitation from cardiac arrest following spinal anaesthesia

open access: yesIndian Journal of Anaesthesia, 2014
Cardiac arrest associated with spinal anaesthesia has been well researched. Myocardial stunning after successful resuscitation from cardiac arrest is seen in up to 2/3 rd of in-hospital cardiac arrests.
Pranjali Madhav Kurhekar   +3 more
doaj   +1 more source

Stress Cardiomyopathy (Takotsubo Cardiomyopathy)

open access: yesИнновационная медицина Кубани, 2023
In this article we describe a rare clinical condition of stress cardiomyopathy (takotsubo cardiomyopathy) for which we, despite controversy and lack of data, identified the main pathogenic pathways (including the direct toxic effect of catecholamines on ...
S. S. Yashin   +2 more
doaj   +1 more source

Connexins and Nitric Oxide Inside and Outside Mitochondria: Significance for Cardiac Protection and Adaptation

open access: yesFrontiers in Physiology, 2018
Irreversible myocardial damage happens in the presence of prolonged and severe ischemia. Several phenomena protect the heart against myocardial infarction and other adverse outcomes of ischemia and reperfusion (IR), namely: hibernation related to stunned
Maria Shvedova   +5 more
doaj   +1 more source

Assessment of Fluid Status in Dialysis: Clinical Importance and Diagnostic Tools [PDF]

open access: yesRevista Portuguesa de Nefrologia e Hipertensão, 2022
Fluid status is crucial for dialysis patients. Volume overload is a major cardiovascular risk factor. Nevertheless, volume depletion is associated with myocardial stunning and cerebral ischaemia.
Juliana Damas, Vasco Fernandes
doaj   +1 more source

Gated metabolic myocardial imaging, a surrogate for dual perfusion-metabolism imaging by positron emission tomography [PDF]

open access: yes, 2017
Acknowledgments The authors are grateful for the help from Dr H Ali and Dr A Dawson. Funding: This study was performed using a research grant from the Aberdeen Royal Hospitals Trust's Endowment Fund, with further support from the Department of Medical ...
AlMohammad, Abdallah   +4 more
core   +1 more source

Brief Myocardial Ischemia Produces Cardiac Troponin I Release and Focal Myocyte Apoptosis in the Absence of Pathological Infarction in Swine

open access: yesJACC: Basic to Translational Science, 2017
Summary: In a porcine model of brief ischemia leading to reversible stunning in the absence of tissue necrosis, we demonstrated delayed release of cardiac troponin I (cTnI) that exceeded the 99th percentile for normal animals 60 min after reperfusion and
Brian R. Weil, PhD   +6 more
doaj   +1 more source

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