Results 251 to 260 of about 103,145 (303)

CHD7 regulates cardiac neural crest cell differentiation through SOX5-mediated self-activation. [PDF]

iScience
Yan S, Bombin A, Liu W, Gonzalez-Giraldo Y, Zhou F, Shi H, Wang Q, Jiao K.   +7 more
europepmc   +1 more source

DNA double-stranded breaks, a hallmark of aging, defined at the nucleotide resolution, are increased and associated with transcription in the cardiac myocytes in LMNA-cardiomyopathy. [PDF]

Cardiovasc Res
Cathcart B, Cheedipudi SM, Rouhi L, Zhao Z, Gurha P, Marian AJ.   +5 more
europepmc   +1 more source

Histopathological Analysis of Myocardial Remodeling Following Heart Failure: A Cadaveric Case Report. [PDF]

Cureus
DeSomma A, Lee JW.
europepmc   +1 more source

Differential bioenergetic profile of human glioblastoma following transplantation of myocyte-derived mitochondria. [PDF]

PLoS One
Marshall KL, Meadows E, Mizener A, Hollander JM, Cifarelli CP.   +4 more
europepmc   +1 more source

Oxidative stress impairs myocyte autophagy, resulting in myocyte hypertrophy [PDF]

Experimental Physiology, 2018
New Findings What is the central question of this study? Does oxidative stress induce impairment of autophagy that results in myocyte hypertrophy early after pressure overload? What is the main finding and its importance? In cultured myocytes, hydrogen peroxide decreased autophagy and increased hypertrophy, and inhibition of autophagy enhanced myocyte
Bao Li, Fu-Zhong Qin
exaly   +3 more sources

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Is the Myofibrillarlytic Myocyte a Forme Fruste Apoptotic Myocyte?

The Annals of Thoracic Surgery, 2005
Myofibrillarlytic (MFL) cells are commonly observed in subendocardial myocardium in myocardial infarction. Because ischemic damage to myocytes is also known to induce apoptosis, we evaluated the prevalence of apoptosis in MFL cells in nine ischemic cardiomyopathic hearts explanted during transplantation.Myocytes with partial or complete clearing of ...
Navneet, Narula, Jagat, Narula, Paul J, Zhang, Nezam, Haider, Puthiyaveettil N, Raghunath, Robin, Brittin, Joseph H, Gorman, Robert C, Gorman, John E, Tomaszewski   +8 more
openaire   +2 more sources

Mechanotransduction in Cardiac Myocytes

Annals of the New York Academy of Sciences, 2004
Abstract: Cardiac myocytes react to diverse mechanical demands with a multitude of transient and long‐term responses to normalize the cellular mechanical environment. Several stretch‐activated signaling pathways have been identified, most prominently guanine nucleotide binding proteins (G‐proteins), mitogen‐activated protein kinases (MAPK), Janus ...
Jan, Lammerding, Roger D, Kamm, Richard T, Lee   +2 more
openaire   +2 more sources

Myocyte Adrenoceptor Signaling Pathways

Science, 2003
Adrenoceptors (ARs), members of the G protein–coupled receptor superfamily, form the interface between the sympathetic nervous system and the cardiovascular system, with integral roles in the rapid regulation of myocardial function. However, in heart failure, chronic catecholamine stimulation of adrenoceptors has been linked to pathologic cardiac ...
Yang, Xiang, Brian K, Kobilka
openaire   +2 more sources

Myocyte injury by hemin

In Vitro Cellular & Developmental Biology - Animal, 1993
Effects of free hemin on myocardium were investigated using a model of neonatal myocyte primary cultures. Cells were subjected to free hemin at concentrations up to 20 microM and equilibrated for 5 h at 37 degrees C. Distribution of hemin in media, cell sarcolemma, and cell interior was evaluated.
V, Bhoite-Solomon, G, Kessler-Icekson, N, Shaklai   +2 more
openaire   +2 more sources