Results 251 to 260 of about 424,484 (404)

Shorter Telomeres and Faster Telomere Attrition in Individuals With Five Syndromic Forms of Intellectual Disability: A Systematic Review and Meta‐Analysis

open access: yesJournal of Intellectual Disability Research, EarlyView.
ABSTRACT Background People with intellectual disability suffer complex challenges due to adaptive functioning limitations, high rates of chronic diseases and shortened lifespans compared with the general population. Telomere shortening is a hallmark of ageing, and short telomeres are linked to neurological disorders.
Sarah M. Hanley   +3 more
wiley   +1 more source

Post-translational processing of rac p21s is important both for their interaction with the GDP/GTP exchange proteins and for their activation of NADPH oxidase.

open access: hybrid, 1992
Seiji Ando   +9 more
openalex   +1 more source

In vitro Activation of the NADPH Oxidase by Fluoride [PDF]

open access: bronze, 1996
Jutta Wölfl   +4 more
openalex   +1 more source

Selective changes in vasopressin neurons and astrocytes in the suprachiasmatic nucleus of Prader–Willi syndrome subjects

open access: yesJournal of Neuroendocrinology, Volume 37, Issue 5, May 2025.
Abstract The hypothalamic suprachiasmatic nucleus (SCN) hosts the central circadian pacemaker and regulates daily rhythms in physiology and behavior. The SCN is composed of peptidergic neuron populations expressing arginine vasopressin (AVP) and vasoactive intestinal polypeptide (VIP), as well as glial cells.
Felipe Correa‐da‐Silva   +13 more
wiley   +1 more source

NADPH oxidases and oxidase crosstalk in cardiovascular diseases: novel therapeutic targets

open access: yesNature Reviews Cardiology, 2019
Yixuan Zhang   +3 more
semanticscholar   +1 more source

A Role for High Mobility Group Box 1 (HMGB1) Release in the Pathogenesis of Gastroesophageal Reflux Disease

open access: yesNeurogastroenterology &Motility, EarlyView.
Incubation of esophageal epithelial cells with a weakly acidic (pH 5) bile salt (deoxycholic acid) induces translocation of HMGB1 from the nucleus to the cytosol, and its subsequent release from the cell. This process can be ameliorated by pre‐treatment with the antioxidant, curcumin.
Tom Leech   +4 more
wiley   +1 more source

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