Results 161 to 170 of about 43,311 (200)
Cell Death in Neurodegenerative Diseases: Molecular Mechanisms and Therapeutic Targets. [PDF]
MedComm (2020)Li T, Zhang Q, Wu Y, Huang R.
europepmc +1 more source
Microvascular autophagy and caspase-3 activation are central regulators of renal fibrosis after ischemia-reperfusion. [PDF]
JCI InsightKim H +9 more
europepmc +1 more source
Zn<sup>2+</sup>/Ca<sup>2+</sup> co-overloaded nano-modulator amplifies reactive oxygen species to potentiate shikonin-induced necroptosis against triple‑negative breast cancer. [PDF]
J NanobiotechnologyXu J +8 more
europepmc +1 more source
Cross-talk among novel programmed cell death pathways: a decisive network in renal ischemia-reperfusion injury. [PDF]
Front ImmunolLou Y +6 more
europepmc +1 more source
Necroptosis molecular mechanisms: Recent findings regarding novel necroptosis regulators [PDF]
Experimental and Molecular Medicine, 2021 Necroptosis is a form of programmed necrosis that is mediated by various cytokines and pattern recognition receptors (PRRs). Cells dying by necroptosis show necrotic phenotypes, including swelling and membrane rupture, and release damage-associated ...
Jaewhan Song, Jinho Seo, Young Woo Nam
exaly +4 more sources
Necroptosis in health and diseases [PDF]
Seminars in Cell and Developmental Biology, 2014 Necroptosis is a form of regulated necrosis that can be activated by ligands of death receptors and stimuli that induce the expression of death receptor ligands under apoptotic deficient conditions. Activation of necroptosis by ligands of death receptors
Junying Yuan
exaly +4 more sources
Trends in Cancer, 2017 Necroptosis is a programmed lytic cell death pathway, deregulation of which is linked to various inflammatory disorders. Escape from programmed cell death and inflammation play a significant role in cancer, and therefore, investigating the role of necroptosis in cancer has been of high interest.
Ayaz Najafov +2 more
exaly +3 more sources
Some of the next articles are maybe not open access.
Related searches:
Related searches:
Journal of Cell Science, 2022
ABSTRACT Necroptosis, or programmed necrosis, is an inflammatory form of cell death with important functions in host defense against pathogens and tissue homeostasis. The four cytosolic receptor-interacting protein kinase homotypic interaction motif (RHIM)-containing adaptor proteins RIPK1, RIPK3, TRIF (also known as TICAM1) and ZBP1 ...
Kidong Kang +2 more
openaire +2 more sources
ABSTRACT Necroptosis, or programmed necrosis, is an inflammatory form of cell death with important functions in host defense against pathogens and tissue homeostasis. The four cytosolic receptor-interacting protein kinase homotypic interaction motif (RHIM)-containing adaptor proteins RIPK1, RIPK3, TRIF (also known as TICAM1) and ZBP1 ...
Kidong Kang +2 more
openaire +2 more sources
Necroptosis in atherosclerosis
Clinica Chimica Acta, 2022Atherosclerosis, a chronic inflammatory disease, is a leading cause of death worldwide. Vascular endothelial cells (VECs), vascular smooth muscle cells (VSMCs) and macrophages play extremely vital roles in the formation of atherosclerotic plaques and subsequent atherosclerosis.
Xiaofan, Zhang +3 more
openaire +2 more sources
Necroptosis in the pathophysiology of preeclampsia
Cell Cycle, 2023Necroptosis is a newly-identified form of gene-regulated cell necrosis that is increasingly considered to be a pathway associated with human pathophysiological conditions. Cells undergoing necroptosis exhibit necrotic phenotypes, including disruption of the plasma membrane integrity, organelle swelling, and cytolysis.
Hongbiao, Yu, Ling, Chen, Boyu, Du
openaire +2 more sources