Results 111 to 120 of about 53,664 (239)

Long‐term outcomes and repeat percutaneous cryoablation in hereditary RCC: a EuRECA cohort

open access: yes
BJU International, EarlyView.
Jens Borgbjerg   +16 more
wiley   +1 more source

Leptin–cholesterol crosstalk in pregnancy: Mechanistic implications for placental function and cardiometabolic programming

open access: yesExperimental Physiology, EarlyView.
Abstract Pregnancy requires the coordinated maternal reorganization of lipid and endocrine pathways to sustain fetal growth and anticipate neonatal nutrition. Leptin, produced by adipose tissue and the placenta, acts as an integrative signal linking maternal energy status with trophoblast function and vascular adaptation, while cholesterol provides ...
Julio Flores   +4 more
wiley   +1 more source

Mapping the Nephron Exercise Incorporates Multiple Learning Strategies

open access: yes, 2017
Introduction: Understanding the location and action of nephron transporters and channels is important to the understanding of renal function. As each region of the nephron is unique in its inclusion of specific transporters and channels, mapping of ...
Lipp, Sarah N.   +5 more
core   +1 more source

Maternal obesity during lactation as a hypertensive cardiovascular risk factor in the offspring

open access: yesExperimental Physiology, EarlyView.
Abstract Maternal obesity is a major public health concern, with growing evidence supporting its role in the developmental programming of cardiometabolic disease. Although prenatal mechanisms have been investigated extensively, the contribution of lactation as a postnatal window of cardiovascular (CV) programming remains insufficiently understood ...
Gabriela Arenas   +2 more
wiley   +1 more source

Changing Protein Permeability with Nephron Loss; Evidence for a Human Remnant Nephron Effect [PDF]

open access: yes
Background If loss of functioning nephrons predisposes to glomerular barotrauma (a ‘remnant nephron’ effect) then glomerular permeability should increase as glomerular filtration rate (GFR) falls, as is observed in animal models of nephron loss.
Odudu A   +5 more
core   +2 more sources

Response to “Methodological Considerations in Interpreting Renal [18F]FDG‐PET After Short‐Term Dapagliflozin Treatment”

open access: yes
Diabetes, Obesity and Metabolism, EarlyView.
Shawn Gugliandolo   +3 more
wiley   +1 more source

Early‐stage 2K1C renovascular hypertension does not increase global MHC‐II or F4/80 abundance in the non‐clipped kidney but reveals focal F4/80+ induction

open access: yesExperimental Physiology, EarlyView.
Abstract The two‐kidney, one‐clip (2K1C) model produces renovascular hypertension. The effects on the clipped kidney (CK) are well described, while the non‐clipped kidney (NCK) is used as a model to evaluate the effects of high blood pressure. Although most of these effects have been described at later stages of 2K1C hypertension, it remains unclear ...
Pilar Cárdenas   +11 more
wiley   +1 more source

Nephron and Podocyte Metrics in Adolescents With a History of Low Birth Weight: A Three-Case Report

open access: yesKidney Medicine
Low birth weight (LBW), which is indicative of impaired fetal growth, is associated with the development of both nephrons and podocytes, increasing the risk of kidney disease later in life. However, the clinical evidence remains limited.
Yuya Yamaguchi   +8 more
doaj   +1 more source

Site of formation of kinins in the dog nephron

open access: yes, 1978
The site of formation of kinins in the nephron was determined by stop-flow studies in dogs. Klinin, inulin, sodium, potassium concentrations were measured in the fractions collected during the stop-flow procedures.
O. A. Carretero   +2 more
core   +1 more source

Born early, age fast: Consequences of premature birth on chronic disease and accelerated ageing

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend ELGANs are exposed to several postnatal pro‐oxidant stressors, including ambient and supplemental oxygen, mechanical ventilation, infections, hyperalimentation, excessive glucocorticoids and intermittent hypoxia. Since endogenous antioxidant defences are underdeveloped, this imbalance promotes oxidative stress and inflammation ...
Estelle B. Gauda   +5 more
wiley   +1 more source

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