Results 121 to 130 of about 178,215 (170)
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Coexisting Membranous Nephropathy and IgA Nephropathy
Fetal and Pediatric Pathology, 2015We herein present a case of a 14-year-old boy with the histological features of coexisting membranous nephropathy (MN) and IgA nephropathy (IgAN). Asymptomatic hematuria/proteinuria was initially detected in school urinary screening, with treatments including oral corticosteroids leading to complete remission.
Masashi, Nishida +2 more
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Combined membranous nephropathy and IgA nephropathy
American Journal of Kidney Diseases, 1998IgA nephropathy (IgAN) and membranous nephropathy (MN) are both common renal biopsy findings that rarely have been described together in the same patient. The significance of this finding is not clear. We present the clinical and pathological data of four patients with combined MN-IgAN and discuss possible pathogenetic mechanisms.
M B, Stokes, C E, Alpers
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COEXISTENCE OF IgA NEPHROPATHY AND MEMBRANOUS NEPHROPATHY
Acta Pathologica Japonica, 1985Renal biopsy findings of a 27‐year‐old female with asymptomatic proteinuria and hematuria revealed two distinct glomerular alterations compatible with both IgA nephropathy and membranous nephropathy. The concomitant presence of two different primary glomerular diseases is very rare.
Y, Kobayashi +3 more
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Critical Care Clinics, 1987
The general topic of kidney damage produced by drugs and toxins is introduced, emphasizing its clinical importance in critically ill patients. Specific toxins are then examined in detail with respect to the mechanisms of toxicity, clinical features, and management.
J H, Sondheimer, S D, Migdal
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The general topic of kidney damage produced by drugs and toxins is introduced, emphasizing its clinical importance in critically ill patients. Specific toxins are then examined in detail with respect to the mechanisms of toxicity, clinical features, and management.
J H, Sondheimer, S D, Migdal
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Annual Review of Medicine, 1987
IgA nephropathy is a syndrome and probably the most important glomerular disease in terms of incidence and mortality. This review documents the clinical and pathological features and concentrates on the evolving thoughts regarding pathogenesis and treatment.
A R, Clarkson +4 more
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IgA nephropathy is a syndrome and probably the most important glomerular disease in terms of incidence and mortality. This review documents the clinical and pathological features and concentrates on the evolving thoughts regarding pathogenesis and treatment.
A R, Clarkson +4 more
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Pediatric Clinics of North America, 1984
It is estimated that about 50 per cent of all patients with Type I diabetes mellitus develop uremia during the course of their disease. Glomerular microvasculopathy is the most serious and predictable threat to longevity. Following a discussion of this disorder, the author outlines a plan for overall management of each phase of kidney disease so that ...
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It is estimated that about 50 per cent of all patients with Type I diabetes mellitus develop uremia during the course of their disease. Glomerular microvasculopathy is the most serious and predictable threat to longevity. Following a discussion of this disorder, the author outlines a plan for overall management of each phase of kidney disease so that ...
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Current Opinion in Pediatrics, 1997
A variety of medications can adversely affect the kidney. Awareness of these potential nephrotoxic effects is crucial to prevention of serious sequelae. This review discusses renal injury caused by the chemotherapeutic agent ifosfamide, the problem of analgesic nephropathy, and the newly identified complication of trimethoprim-induced hyperkalemia.
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A variety of medications can adversely affect the kidney. Awareness of these potential nephrotoxic effects is crucial to prevention of serious sequelae. This review discusses renal injury caused by the chemotherapeutic agent ifosfamide, the problem of analgesic nephropathy, and the newly identified complication of trimethoprim-induced hyperkalemia.
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Seminars in Nephrology, 2003
Renal involvement is common in leptospirosis. Clinical manifestations vary from urinary sediment changes to acute renal failure. Renal failure is observed in 44% to 67% of patients. Hypokalemia frequently occurs. Severe hypotension is an important warning sign for the later development of renal and pulmonary complications.
Visith, Sitprija +2 more
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Renal involvement is common in leptospirosis. Clinical manifestations vary from urinary sediment changes to acute renal failure. Renal failure is observed in 44% to 67% of patients. Hypokalemia frequently occurs. Severe hypotension is an important warning sign for the later development of renal and pulmonary complications.
Visith, Sitprija +2 more
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Clinical Nephrology, 2015
Balkan endemic nephropathy (BN), frequently associated to upper urothelial cancer, is a familial chronic tubulointerstitial disease with insidious onset and slow progression to end-stage renal disease. After 60 years of research, its cause remains the major unanswered question.
Vladisav, Stefanovic +2 more
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Balkan endemic nephropathy (BN), frequently associated to upper urothelial cancer, is a familial chronic tubulointerstitial disease with insidious onset and slow progression to end-stage renal disease. After 60 years of research, its cause remains the major unanswered question.
Vladisav, Stefanovic +2 more
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Australian and New Zealand Journal of Medicine, 1983
Abstract:Recent reports have suggested that there is a specific form of mesangial proliferative glomerulonephritis (GN) characterised by a diffuse and generalised mesangial deposition of IgM. The study included 149 consecutive patients with primary GN, who had adequate renal tissue for detailed light and immunofluorescent microscopy. Forty of these 149
M J, Murphy, R R, Bailey, A R, McGiven
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Abstract:Recent reports have suggested that there is a specific form of mesangial proliferative glomerulonephritis (GN) characterised by a diffuse and generalised mesangial deposition of IgM. The study included 149 consecutive patients with primary GN, who had adequate renal tissue for detailed light and immunofluorescent microscopy. Forty of these 149
M J, Murphy, R R, Bailey, A R, McGiven
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