Results 261 to 270 of about 216,217 (284)
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2003
Abstract It is increasingly imperative that Alzheimer’s disease (AD) be detected as early in its clinical course as possible. The goals of treatment of AD are to slow the progress and control the symptoms, thus increasing the period of relatively preserved function while decreasing the period of disability.
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Abstract It is increasingly imperative that Alzheimer’s disease (AD) be detected as early in its clinical course as possible. The goals of treatment of AD are to slow the progress and control the symptoms, thus increasing the period of relatively preserved function while decreasing the period of disability.
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Neuropsychiatric Symptoms of Hepatitis C
Issues in Mental Health Nursing, 2008More than 4 million (2%) people in the United States have been infected with the hepatitis C virus, of whom 2.7 million are chronically infected. The current treatment for chronic hepatitis C patients is Interferon and ribavirin combination therapy, which is associated with numerous neuropsychiatric side effects. The most common are fatigue, depression,
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Neuropsychiatric Symptoms and Tick-Borne Diseases
2022In North America, Lyme disease (LD) is primarily caused by the spirochetal bacterium Borrelia burgdorferi, transmitted to humans by Ixodes species tick bites, at an estimated rate of 476,000 patients diagnosed per year. Acute LD often manifests with flu-like symptoms and an expanding rash known as erythema migrans (EM) and less often with neurologic ...
Shannon L, Delaney +2 more
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Management of neuropsychiatric symptoms in dementia
Current Opinion in Neurology, 2023Purpose of review The purpose is to review the results and clinical implications of recent studies of neuropathology in relation to neuropsychiatric symptoms (NPS) in Alzheimer's disease and related dementias, and discuss new therapeutic approaches based on evidence from clinical trials. Recent
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δ scores predict multiple neuropsychiatric symptoms
International Journal of Geriatric Psychiatry, 2020AbstractObjectivesDementia severity is strongly related to Spearman's general intelligence factor “g”, via the latent dementia phenotype “δ” and is distinct from domain‐specific cognitive impairments arising from disease‐specific regional pathologies.
Donald R. Royall, Raymond F. Palmer
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Neuropsychiatric Symptoms in Dementia
ContinuumThis article discusses the prevalence, pathophysiology, assessment, and management of neuropsychiatric symptoms in patients with dementia.There is a growing body of evidence localizing neuropsychiatric symptoms in dementia to frontal circuits in the brain, as well as relating them to pathologic changes seen in different dementias.
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Neuropsychiatric Symptoms of CADASIL
2013CADASIL is an inherited small artery disease caused by mutations of the NOTCH3 gene. The disease is responsible for migraine with aura at onset, for transient ischemic attacks and stroke during mid-adulthood, and can lead progressively to dementia. The neuropsychiatric manifestations of CADASIL include mood and behavior disturbances and various degrees
Hugues Chabriat, Sonia Reyes
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Neuropsychiatric Symptom Assessments in Toxic Exposure
Psychiatric Clinics of North America, 2013The goal of this review is to provide guidelines for evaluating psychiatric and mood changes that result from neurotoxicity. Mood changes that are often seen to varying degrees in neurotoxicity include increased anxiety, depression, irritability, impulsiveness, and psychosis. Some common agents that induce neurotoxicity include drugs, heavy metals, and
Lisa H, Mason +2 more
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Post-stroke Neuropsychiatric Symptoms
2018The term ‘neuropsychiatric symptoms’ (NPS) collectively identify the following behavioral symptoms; psychosis, apathy, depression, difficulty with sleeping, aggression, agitation and disordered eating. Literature suggests that there is a high prevalence of these symptoms experienced by stroke survivors.
B. I. Buijck, T. Silveira
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The Elusive Neuropsychiatric Symptoms
1984In the second Salmon lecture, presented at the New York Academy of Medicine, Michael Rutter (1982) reviews the overwhelming evidence that “it is possible for overt and indisputable brain damage to occur, and yet for a careful clinical examination to reveal no definite (neurological) abnormalities” (p. 22).
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