Results 1 to 10 of about 32,471 (163)
Newcastle Disease Virus Infection in Quail [PDF]
Veterinary Pathology, 2018 Newcastle disease (ND), caused by virulent strains of Newcastle disease virus (NDV), is a devastating disease of poultry worldwide. The pathogenesis of ND in quail is poorly documented. To characterize the ability of virulent NDV strains to replicate and cause disease in quail, groups of 14 two-week-old Japanese quail ( Coturnix ...
Leonardo Susta +2 more
exaly +3 more sources
STUDIES ON NEWCASTLE DISEASE VIRUS [PDF]
Journal of Experimental Medicine, 1948 The application of the 50 per cent embryo mortality to a study of the virus of Newcastle is described. It has been evaluated by a series of duplicate titrations of the same sample of virus. In seven such titrations the largest difference between the two was 10–0.4. It is therefore believed that a difference of 0.6 log is probably significant and of 1.0
F B Bang
exaly +4 more sources
Newcastle disease virus pathotypes [PDF]
Avian Pathology, 1974 The clinical signs, mortality and postmortem findings following infection of six-week-old chicks with nine strains of Newcastle disease virus were studied. Although strains could be divided into four pathotypes the divisions were not clear-cut. The most prominent feature of disease following infection by two isolates from the post-1970 USA epidemic, a ...
D J, Alexander, W H, Allan
openaire +2 more sources
c‐Rel–dependent Chk2 signaling regulates the DNA damage response limiting hepatocarcinogenesis
Hepatology, EarlyView., 2022 In response to genotoxic injury, c‐Rel upregulates ATM‐Chk2‐p53 pathway DNA damage proteins to limiting hepatocarcinogenesis. Abstract Background and Aims Hepatocellular carcinoma (HCC) is a leading cause of cancer‐related death. The NF‐κB transcription factor family subunit c‐Rel is typically protumorigenic; however, it has recently been reported as a
Jack Leslie +17 more
wiley +1 more source
Transcriptional map for Newcastle disease virus [PDF]
Journal of Virology, 1980 A transcriptional map of Newcastle disease virus was determined by measuring the kinetics of UV inactivation of the transcription of individual genes and of viral infectivity. The inactivation of single genes was monitored by measuring the reduction in the accumulation of viral gene products in vivo and in vitro.
P L, Collins, L E, Hightower, L A, Ball
openaire +2 more sources
Strain differences in the Newcastle disease virus [PDF]
Journal of Hygiene, 1956 The biological characters of eight strains of the Newcastle disease virus, isolated in different parts of the world between 1933 and 1951, have been studied and compared in detail.Two types of the virus have been distinguished, one from the Newcastle disease prevalent in Europe and Australia, the other from that occurring in the United States of ...
L W, MACPHERSON, R H, SWAIN
openaire +2 more sources
Reverse Genetics of Newcastle Disease Virus [PDF]
, 2017 Reverse genetics allows for the generation of recombinant viruses or vectors used in functional studies, vaccine development, and gene therapy. This technique enables genetic manipulation and cloning of viral genomes, gene mutation through site-directed mutagenesis, along with gene insertion or deletion, among other studies. An in vitro infection-based
Cardenas-Garcia, Stivalis +1 more
openaire +2 more sources
Precursor Protein for Newcastle Disease Virus [PDF]
Journal of Virology, 1973 The course of viral protein synthesis during infection of chicken embryo fibroblasts with Newcastle disease virus (NDV) L. Kansas has been followed by using sodium dodecyl sulfate polyacrylamide gel electrophoresis. Of the three major virion polypeptide molecular weight classes, I (78,400 daltons), II (53,500 daltons), and III (37,600 daltons), only II,
A C, Samson, C F, Fox
openaire +2 more sources
Noncytopathic mutants of Newcastle disease virus [PDF]
Journal of Virology, 1978 We have isolated a novel class of mutants of Newcastle disease virus which are less cytopathic than their virulent parent but are still capable of infectious virus production. Unlike wild-type virus, the mutants did not form plaques after 2 days of incubation; they did, however, make hemadsorbing spots.
C H, Madansky, M A, Bratt
openaire +2 more sources
Thermal Inactivation of Newcastle Disease Virus [PDF]
Applied Microbiology, 1970 The rate of destruction of hemagglutinins and infectivity of Newcastle disease virus was determined over a temperature range of 37.8 to 60 C. From the calculated values of δH and δS, it was concluded that inactivation of the hemagglutinating activity and viral infectivity was due to protein denaturation.
G A, DiGioia +3 more
openaire +2 more sources