Results 221 to 230 of about 29,480 (254)

Pharmacological inhibition of the PERK pathway modulates hepatocellular carcinoma growth and immune signaling

FEBS Open Bio, EarlyView.
Pharmacological inhibition of PERK in a DEN‐induced mouse model of liver cancer does not reduce tumor burden but alters cellular stress signaling. Despite blocking PERK activity, downstream stress responses, including CHOP expression, remain active, suggesting compensatory mechanisms within the unfolded protein response that may influence tumor ...
Ada Lerma‐Clavero, Maria Kopsida, Nathalie Arendt, Hans Lennernäs, Markus Sjöblom, Femke Heindryckx   +5 more
wiley   +1 more source

UNICORN and the Evolution of Coronary Protection in the Redo-TAVR Era. [PDF]

JACC Asia
Song GY.
europepmc   +1 more source

Treatment of oligometastatic lung cancers: new data, new drivers, new approaches. [PDF]

BMJ Oncol
O'Dwyer N, Palma DA.
europepmc   +1 more source

Carotid Artery Stenting: Evolution, Evidence, and Contemporary Practice in the Era of Intensive Medical Therapy. [PDF]

Life (Basel)
Dixit S, Anamika F, Multani A, Rosenzveig A, Kathirvel B, Degala S, Thalamati M, Kirksey L, Bajzer C, Raskin D, Nanjundappa A.   +10 more
europepmc   +1 more source

Bibliometric and trend analysis of the top 100 most-cited articles on anterior lumbar interbody fusion (ALIF). [PDF]

Brain Spine
Hafiz B, Alsharif T, Hamzah A, Sukkar F, Bassi Y, Alghamdi M, Tariq M, Bokhari R, Baeesa S.   +8 more
europepmc   +1 more source

Reconfiguring agency under platform governance: Baby Boomer and Generation X singers negotiating identity, algorithms, and monetization. [PDF]

Front Sociol
Hastuti A, Widodo S, Suhariadi F.
europepmc   +1 more source

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Newer developments in idiopathic pulmonary fibrosis in the era of anti-fibrotic medications

Expert Review of Respiratory Medicine, 2016
Idiopathic pulmonary fibrosis (IPF) is the most common interstitial lung disease with a fatal prognosis. Over the last decade, the concepts in pathobiology of pulmonary fibrosis have shifted from a model of chronic inflammation to dysregulated fibroproliferative repair in genetically predisposed patients.
Ganesh Raghu
exaly   +3 more sources