Results 171 to 180 of about 78,616 (322)

Primary structure of a developmentally regulated nicotinic acetylcholine receptor protein from Drosophila [PDF]

, 1986
Irm Hermans‐Borgmeyer, Dieter Zopf, Rolf -Peter Ryseck, Bernd Hovemann, Heinrich Betz, Eckart D. Gundelfinger   +5 more
openalex   +1 more source

Nonsteroidal Anti‐Inflammatory Drugs as Modulators of Cation Channels: Fenamates Repurposing in Channelopathies

ChemMedChem, Volume 20, Issue 20, October 15, 2025.
Beyond their established role as nonsteroidal anti‐inflammatory drugs (NSAIDs), fenamates (niflumic, flufenamic, mefenamic, meclofenamic, and tolfenamic acids) have recently been identified as modulators of cationic ion channels. This review highlights their differential effects on ion channel activity and explores their potential for repurposing in ...
Paola Laghetti, Concetta Altamura, Simone Dell’Atti, Jean‐François Desaphy, Ilaria Saltarella   +4 more
wiley   +1 more source

5‐Hydroxytryptamine decreases the sensitivity of nicotinic acetylcholine receptor in bull‐frog sympathetic ganglion cells.

, 1986
T. Akasu, K. Koketsu
openalex   +2 more sources

Filamented Light (FLight) Bioprinting of Mini‐Muscles with Self‐Renewal Potential

Advanced Materials, Volume 37, Issue 40, October 9, 2025.
This work presents the FLight biofabrication for engineering in vitro muscle constructs by photocrosslinking pristine collagen and fibrinogen using ruthenium. The resulting mini‐muscles retain in vivo‐like tissue organization and a Pax7⁺ cell pool.
Hao Liu, Michael Winkelbauer, Jakub Janiak, Patrick Weber, Ali Kerem Kalkan, Inseon Kim, Parth Chansoria, Ori Bar‐Nur, Marcy Zenobi‐Wong   +8 more
wiley   +1 more source

Modeling presynaptic inhibition by the amyloid precursor protein demonstrates one potential mechanism for preventing runaway synaptic modification in Alzheimer's disease

Alzheimer's &Dementia, Volume 21, Issue 10, October 2025.
Abstract INTRODUCTION Previous simulations of Hebbian associative memory models inspired the malignant synaptic growth hypothesis of Alzheimer's disease (AD), which suggests that cognitive impairments arise due to runaway synaptic modification resulting from poor separation between encoding and retrieval.
Dylan Barber, Michael E. Hasselmo, Heather C. Rice   +2 more
wiley   +1 more source

Formation of the alpha-bungarotoxin binding site and assembly of the nicotinic acetylcholine receptor subunits occur in the endoplasmic reticulum.

, 1987
McHardy M. Smith, Jon Lindstrom, John P. Merlie   +2 more
openalex   +1 more source

Functional expression of nicotinic acetylcholine receptors containing rat α7 subunits in human SH‐SY5Y neuroblastoma cells [PDF]

, 1994
Elzbieta Puchacz, Bruno Buisson, Daniel Bertrand, Ronald J. Lukas   +3 more
openalex   +1 more source

Entwined processes in Alzheimer's disease: Brain cellular senescence and Alzheimer's disease pathology

Alzheimer's &Dementia, Volume 21, Issue 10, October 2025.
Abstract Alzheimer's disease (AD) is one of the most common age‐related neurodegenerative diseases with severe progressive dementia. Amyloid beta (Aβ) deposition and phosphorylated tau accumulation are two typical AD pathologies, which could drive senescence in various types of brain cells.
Wen Li, Ying Han, Peichang Wang, Qiao Song   +3 more
wiley   +1 more source

Determination of the sites of cAMP-dependent phosphorylation on the nicotinic acetylcholine receptor.

, 1987
GH Yee, RL Huganir
openalex   +1 more source

Nicotinic acetylcholine receptors in porcine hypophyseal intermediate lobe cells. [PDF]

, 1990
Zhongwei Zhang, P. Feltz
openalex   +1 more source