A Scoping Review of Pathogenesis, Current Treatments, and Novel Approaches for Vitiligo
ABSTRACT Background Vitiligo is a chronic skin disorder characterized by loss of melanocytes, causing depigmentation and psychological distress. Its pathogenesis is complex and multifactorial, involving genetic predisposition, autoimmune, oxidative stress, and neurogenic mechanisms.
Nusaibah Sallehuddin +3 more
wiley +1 more source
The I-Abm12 mutation, which confers resistance to experimental myasthenia gravis, drastically affects the epitope repertoire of murine CD4+ cells sensitized to nicotinic acetylcholine receptor. [PDF]
Matteo Bellone +4 more
openalex +1 more source
ABSTRACT Background Acne vulgaris (AV) is a common inflammatory skin disease during adolescence. Metformin (MET) has recently been found to have the effects of regulating lipid disorders, suggesting its potential benefits in the treatment of AV patients. Method Recruited 18 patients with moderate to severe AV, and then received MET treatment (AVM group)
Jie Hu +5 more
wiley +1 more source
Effects of Selective α7 Nicotinic Acetylcholine Receptor Stimulation in Oligodendrocytes: Putative Implication in Neuroinflammation. [PDF]
Guerriero C +6 more
europepmc +1 more source
Nicotinic acetylcholine receptor signaling maintains epithelial barrier integrity. [PDF]
Katheder NS +11 more
europepmc +1 more source
Autoantibodies and Inflammation in Schizophrenia
The potential role of autoantibodies in the etiology of schizophrenia is a key research focus because growing evidence suggests an association between immune dysfunction and psychotic disorders. This hypothesis is supported by findings indicating immune‐related abnormalities in patients with schizophrenia, including chronic inflammation and genetic ...
Hiroki Shiwaku
wiley +1 more source
AGONIST-INDUCED AFFINITY ALTERATIONS OF A CENTRAL NERVOUS SYSTEM NICOTINIC ACETYLCHOLINE RECEPTOR
Ronald J Lukasiewicz, Edward L Bennett
openalex +1 more source
Activation of the α7 nicotinic acetylcholine receptor mitigates cognitive deficits in mice with sepsis-associated encephalopathy by inhibiting microglial pyroptosis. [PDF]
Wang Q +7 more
europepmc +1 more source

