Results 241 to 250 of about 172,466 (303)

Context-dependent roles of 11β-HSD1 in bone and skeletal muscle diseases. [PDF]

Am J Transl Res
Huo L, Sui W, Wang S, Zhu W, Yang Y, Zhang X, Zhang Y, Wang X.   +7 more
europepmc   +1 more source

Anoctamin 5 mutation leads to abnormal bone homeostasis of GDD by regulating AMPK-dependent glucose metabolism. [PDF]

Front Endocrinol (Lausanne)
Li H, Xu H, Sun K, Wang S, Zhang M, Lin L, Zhang S, Dong R, Hu Y.   +8 more
europepmc   +1 more source

Traditional Chinese Medicine for osteoporosis management: from molecular mechanisms to drug discovery. [PDF]

Chin Med
Yan S, Du Q, Zhao L, Liu Q, Wu M, Xu Z, Shen J.   +6 more
europepmc   +1 more source

Osteoblastic metastases

, 2011
openaire   +1 more source

mTOR signalling mediates the spinal osteoblast pathotype at the curve apex in adolescent idiopathic scoliosis

Northall EH, Nicholson TA, Hazeldine J, Grover LM, McGettrick HM, Ede MN, Naylor AJ, Jones SW.   +7 more
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Osteoclast–osteoblast communication

Archives of Biochemistry and Biophysics, 2008
Cells in osteoclast and osteoblast lineages communicate with each other through cell-cell contact, diffusible paracrine factors and cell-bone matrix interaction. Osteoclast-osteoblast communication occurs in a basic multicellular unit (BMU) at the initiation, transition and termination phases of bone remodeling.
Koichi, Matsuo, Naoko, Irie
openaire   +2 more sources

Pinacidil stimulates osteoblast function in osteoblastic MC3T3-E1 cells

Immunopharmacology and Immunotoxicology, 2013
This study examined the effect of pinacidil, a nonselective adenosine triphosphate-sensitive potassium channel opener, on the function of osteoblastic MC3T3-E1 cells. Pinacidil caused a significant elevation of collagen synthesis, alkaline phosphatase activity, osteocalcin synthesis and mineralization in the cells (p 
Kwang Sik, Suh, Young Soon, Lee, Eun Mi, Choi   +2 more
openaire   +2 more sources