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Posters, 2017
Kindler syndrome is a rare form of epidermolysis bullosa, which represents a group of hereditary diseases characterised by particular fragility of the skin and mucosa, manifesting in the appearance of bullae in minor traumas. We present the case of a 12-year-old girl who shows clinical elements characteristic of Kindler syndrome.
Dalia Dop +5 more
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Kindler syndrome is a rare form of epidermolysis bullosa, which represents a group of hereditary diseases characterised by particular fragility of the skin and mucosa, manifesting in the appearance of bullae in minor traumas. We present the case of a 12-year-old girl who shows clinical elements characteristic of Kindler syndrome.
Dalia Dop +5 more
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European Journal of Biochemistry, 1998
Human immunodeficiency virus type‐1 (HIV‐1) reverse transcriptase (RT) initiates reverse transcription from tRNALys3. HIV‐1 RT is a heterodimer consisting of two polypeptides, p66 and p51. In this work, the possible role of each subunit of RT in the interaction with its natural primer tRNALys3 was studied. Two recombinant forms of HIV‐1 RT, heterodimer
E, Dufour +7 more
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Human immunodeficiency virus type‐1 (HIV‐1) reverse transcriptase (RT) initiates reverse transcription from tRNALys3. HIV‐1 RT is a heterodimer consisting of two polypeptides, p66 and p51. In this work, the possible role of each subunit of RT in the interaction with its natural primer tRNALys3 was studied. Two recombinant forms of HIV‐1 RT, heterodimer
E, Dufour +7 more
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Absence of p51 mutation in human hepatocellular carcinoma
Cancer Letters, 2000The p51 gene encodes a protein with significant homology to p53. To investigate the involvement of the p51 gene in human hepatocarcinogenesis, mutation analysis of the p51 gene was performed in 54 cases of hepatocellular carcinoma (HCC). No mutations causing amino acid substitutions or frameshifts were found by polymerase chain reaction-single strand ...
K, Hamada +7 more
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Absence of p51 alteration in human ovarian cancer
International Journal of Oncology, 2001The p51 gene encodes a protein with significant homology to p53, thus, it is a candidate tumor suppressor gene. To investigate the involvement of the p51 gene in human ovarian carcinogenesis, p51 gene alterations were examined in primary ovarian cancers and ovarian cancer cell lines. Mutation analysis of the p51 gene was performed in 40 primary ovarian
H, Shinozaki +5 more
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The Prostate, 2001
AbstractBACKGROUNDp73 and p51 are genes possessing amino‐acid similarities to p53. We previously found no mutation in p73 in prostatic carcinoma, but did find abnormal expression of the gene. Involvement of these genes in prostatic carcinogenesis is still poorly understood.METHODSMutation analysis of the p51 gene and allelotyping of 3q28, on which p51 ...
H, Takahashi +8 more
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AbstractBACKGROUNDp73 and p51 are genes possessing amino‐acid similarities to p53. We previously found no mutation in p73 in prostatic carcinoma, but did find abnormal expression of the gene. Involvement of these genes in prostatic carcinogenesis is still poorly understood.METHODSMutation analysis of the p51 gene and allelotyping of 3q28, on which p51 ...
H, Takahashi +8 more
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2023
The Minor Planet Electronic Circulars contain information on unusual minor planets, routine data on comets and natural satellites, and occasional editorial announcements. They are published on behalf of Division F of the International Astronomical Union by the Minor Planet Center, Smithsonian Astrophysical Observatory, Cambridge, MA 02138, U.S.A.
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The Minor Planet Electronic Circulars contain information on unusual minor planets, routine data on comets and natural satellites, and occasional editorial announcements. They are published on behalf of Division F of the International Astronomical Union by the Minor Planet Center, Smithsonian Astrophysical Observatory, Cambridge, MA 02138, U.S.A.
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P51 Two cases of Evans' syndrome in pregnancy
Thrombosis Research, 2009Results: At least one inherited or acquired thrombophilic factor was detected in 66.9% of women with repeated IVF failure. The prevalence for FV Leiden was 10.6%, 5.6% for prothrombin and 38.75% for MTHFR. The other thrombophilic factors were found as (PZ: 10.6%, thrombomodulin: 9.4%), antiphospholipid antibodies in 7.5% of cases. In a control group of
E. Lefkou, C. Nelson-Piercy, B.J. Hunt
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