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Patterns of immune cell infiltration and oxidative stress in cervical cancer. [PDF]
Mlambo A, Su S, Dhlamini Q, Zhang Y.
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Diagnosis, Management, and Follow-Up of Human Papillomavirus Type 16 (HPV-16) Lesions in the Oral Cavity. [PDF]
Patel M +3 more
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New Insights into Pathogenesis and Management of Keratoacanthoma: A Narrative Review. [PDF]
Hyeraci M +13 more
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Actinic Keratosis and Human Papillomaviruses: may their relationship constitute a new approach for actinic keratosis management? [PDF]
De Francesco MA +7 more
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MCPyV small T antigen enhances HPV16 oncogene expression, promotes Ca Ski cell proliferation, and reduces 5-fluorouracil-induced apoptosis in cervical cancer cells. [PDF]
Pakdel T +4 more
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Applied Biochemistry and Biotechnology, 2023
E6 and E7 human papillomavirus (HPV) oncoproteins play a significant role in the malignant transformation of infected cervical cancer cells via suppression of tumour suppressor pathways by targeting p53 and pRb, respectively. This study aimed to investigate the anticancer effects of Oroxylum indicum (OI) leaves' methanol extract on SiHa cervical cancer
Noor Nabilah Talik Sisin +4 more
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E6 and E7 human papillomavirus (HPV) oncoproteins play a significant role in the malignant transformation of infected cervical cancer cells via suppression of tumour suppressor pathways by targeting p53 and pRb, respectively. This study aimed to investigate the anticancer effects of Oroxylum indicum (OI) leaves' methanol extract on SiHa cervical cancer
Noor Nabilah Talik Sisin +4 more
openaire +2 more sources
Cancer Letters, 2004
Approximately 100% of cervical carcinomas are causally linked to infections with high-risk human papillomaviruses (HPVs), whose oncogenicity has been assigned to the continued expression of two early viral genes, E6 and E7. Reversal of the transformed phenotype by inhibiting E6/E7 gene expression therefore provides a suitable goal for tumor therapy. We
Klaus, Braun +7 more
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Approximately 100% of cervical carcinomas are causally linked to infections with high-risk human papillomaviruses (HPVs), whose oncogenicity has been assigned to the continued expression of two early viral genes, E6 and E7. Reversal of the transformed phenotype by inhibiting E6/E7 gene expression therefore provides a suitable goal for tumor therapy. We
Klaus, Braun +7 more
openaire +2 more sources

