Results 151 to 160 of about 15,854 (193)
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Dermatologic Clinics, 1988
The term pemphigus refers to a group of autoimmune intraepidermal blistering diseases of the skin and mucous membranes. Several clinical variants of pemphigus are recognized. The major histologic feature of all variants is acantholysis, the disruption of normal cell-to-cell adhesion, which leads to intraepidermal blister formation.
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The term pemphigus refers to a group of autoimmune intraepidermal blistering diseases of the skin and mucous membranes. Several clinical variants of pemphigus are recognized. The major histologic feature of all variants is acantholysis, the disruption of normal cell-to-cell adhesion, which leads to intraepidermal blister formation.
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Immunotherapy, 2017
Pemphigus is a severe autoimmune blistering disease mediated by pathogenic anti-desmoglein antibodies leading to an inter keratinocyte disjunction. Rituximab is a monoclonal antibody that binds to the CD-20 antigen of B lymphocytes, which causes B-cell depletion and a subsequent reduction in pathogenic autoantibodies.
Hebert, Vivien, Joly, Pascal
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Pemphigus is a severe autoimmune blistering disease mediated by pathogenic anti-desmoglein antibodies leading to an inter keratinocyte disjunction. Rituximab is a monoclonal antibody that binds to the CD-20 antigen of B lymphocytes, which causes B-cell depletion and a subsequent reduction in pathogenic autoantibodies.
Hebert, Vivien, Joly, Pascal
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Pemphigus: Pemphigus vulgaris and pemphigus foliaceus
1990The term pemphigus stems from the Greek pemphix (blister) [1] and describes a group of chronic blistering skin diseases in which autoantibodies are directed against the cell surface of keratinocytes, resulting in the loss of cohesion between epidermal cells, through a process called acantholysis. Pemphigus is divided into pemphigus vulgaris (PV) with a
Stephan Müller, John R. Stanley
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Penicillamine-associated pemphigus: Is it really pemphigus?
Journal of the American Academy of Dermatology, 1981Penicillamine-associated bullous eruptions share with spontaneously occurring pemphigus intraepidermal acantholysis, epidermal intercellular deposition of immunoglobulin, and circulating serum antibody against the intercellular regions of the epidermis.
J L, Troy +3 more
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HERPETIFORM PEMPHIGUS, A VARIABLE PATTERN OF PEMPHIGUS
International Journal of Dermatology, 1975ABSTRACT: A patient with the clinical features of dermatitis herpetiformis and the histological features of pemphigus, subcorneal dermatosis and dermatitis herpetiformis did not respond to treatment with pyridine or sulfones. Systemic steroids and azothioprine were effective. Immunofluorescence studies demonstrated circulating pemphigus antibodies and
S, Jablonska +3 more
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Interventions for pemphigus vulgaris and pemphigus foliaceus
Cochrane Database of Systematic Reviews, 2009A range of interventions have been described for treatment of pemphigus, however the optimal therapeutic strategy has not been established.To assess the efficacy and safety of all interventions used in the management of pemphigus vulgaris and pemphigus foliaceus.We searched the Cochrane Skin Group Specialised Register (October 2008), The Cochrane ...
Linda K Martin +5 more
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International Journal of Dermatology, 1991
Abstract: A family in which two sisters are affected by pemphigus is described. Literature research has revealed the presence of 25 families, comprising 53 patients, in which pemphigus existed in more than one member. The relationships between those affected were mainly parent‐child or sibling‐sibling.
A, Feinstein +3 more
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Abstract: A family in which two sisters are affected by pemphigus is described. Literature research has revealed the presence of 25 families, comprising 53 patients, in which pemphigus existed in more than one member. The relationships between those affected were mainly parent‐child or sibling‐sibling.
A, Feinstein +3 more
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Archives of Dermatological Research, 1982
The reports of induced pemphigus have multiplied over the last 10 years. Several heterogeneous factors, such as drugs (penicillamine, pyritinol, captopril, rifampicin, etc.), physical agents (burns, UV, and ionizing radiation) and viruses, can play an inducing role. Usually, the disease is preceded by prodromal, non-specific lesions.
RUOCCO, Vincenzo, PISANI M.
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The reports of induced pemphigus have multiplied over the last 10 years. Several heterogeneous factors, such as drugs (penicillamine, pyritinol, captopril, rifampicin, etc.), physical agents (burns, UV, and ionizing radiation) and viruses, can play an inducing role. Usually, the disease is preceded by prodromal, non-specific lesions.
RUOCCO, Vincenzo, PISANI M.
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Dermatologica, 2009
In the skin organ culture model of pemphigus, fibronectin concentrations of 300 and 500 μg/ml inhibited pemphigus plasma-induced acantholysis and intraepidermal binding of the pemphigus antibodies examined by direct immunofluorescence. A direct interaction of fibronectin with pemphigus antibodies could not be demonstrated by chromatography of pemphigus
T, Hunziker +2 more
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In the skin organ culture model of pemphigus, fibronectin concentrations of 300 and 500 μg/ml inhibited pemphigus plasma-induced acantholysis and intraepidermal binding of the pemphigus antibodies examined by direct immunofluorescence. A direct interaction of fibronectin with pemphigus antibodies could not be demonstrated by chromatography of pemphigus
T, Hunziker +2 more
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2015
Paraneoplastic pemphigus is a rare but severe autoimmune disease characterized by severe stomatitis and a variety of cutaneous manifestations in association with an underlying neoplasia. Pulmonary involvement may also occur. The pathogenesis involves the production of autoantibodies against desmogleins, plakins, and the protease inhibitor alpha-2 ...
Diercks, G.F.H. +3 more
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Paraneoplastic pemphigus is a rare but severe autoimmune disease characterized by severe stomatitis and a variety of cutaneous manifestations in association with an underlying neoplasia. Pulmonary involvement may also occur. The pathogenesis involves the production of autoantibodies against desmogleins, plakins, and the protease inhibitor alpha-2 ...
Diercks, G.F.H. +3 more
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