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ADAM17 and its proteolytic targets in disease pathogenesis
The FEBS Journal, EarlyView.ADAM17 as a multifunctional sheddase with contrasting roles across inflammatory, metabolic, cardiovascular, and neoplastic diseases. Through regulated activation by iRhom, iTAP/FRMD8, and tetraspanins, ADAM17 cleaves diverse membrane ligands and receptors, thereby promoting inflammation, fibrosis, obesity, insulin resistance, and tumor progression ...
Abdulbasit Amin, Marina Badenes
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Proteolytic remodelling of the extracellular matrix by pericytes
The FEBS Journal, EarlyView.Pericytes are specialised perivascular cells intimately connected with endothelial cells and essential for the maintenance of vascular beds. They contribute to the formation and remodelling of the extracellular matrix by actively secreting proteases and protease inhibitors.
Tina Burkhard +4 more
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Placental insufficiency and postmaturity
European Journal of Obstetrics & Gynecology and Reproductive Biology, 1975Full placental development and functioning are achieved around the 5th month of gestation. Thereafter, until birth, signs of gradual placental aging are observed which are partly compensated by an increase in trophoblastic surface area to maintain adequate fetal oxygen and nutrient supply (Krantz and Kubli, 1967; Hellegers, 1969).
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Elevated circulating and placental SPINT2 is associated with placental insufficiency
Placenta, 2021No description ...
Murphy, Ciara +13 more
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Placental Gene Expression in a Rat ‘Model’ of Placental Insufficiency
Placenta, 2010Placental insufficiency is a major factor associated with pregnancy complications such as miscarriages, intrauterine growth restriction and pre-eclampsia. Recent studies have identified the Brown Norway (BN) rat as a natural 'model' of placental insufficiency associated with decreased trophoblast remodeling of maternal uterine arteries.Genetic pathways
R, Goyal +3 more
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Placental insufficiency and its consequences
European Journal of Obstetrics & Gynecology and Reproductive Biology, 2003Placental insufficiency is a process leading to progressive deterioration in placental function and a decrease in transplacental transfer of oxygen and nutrients to the fetus. The resulting fetal hypoxemia is the major stimulus involved in the reduction in fetal growth as an attempt to reduce metabolic demands by the growing fetus.
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Placental insufficiency and the small-for-dates baby
American Journal of Obstetrics and Gynecology, 1972Abstract A blind prospective pathologic survey of the placenta, designed to detect cases of placental insufficiency, was carried out during 1970, with the use of a scoring system involving over 7 main parameters. An 80 per cent correlation with the pediatric assessment of the baby was obtained. The system was then reassessed statistically, and scores
J M, Scott, J M, Jordan
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Leptin: A Potential Marker of Placental Insufficiency
Gynecologic and Obstetric Investigation, 2003To investigate placental leptin production in placental insufficiency, placental leptin production was measured in women with severe preeclampsia (group 1) and in normotensive pregnancies associated with intrauterine growth restriction (group 2), compared to controls (group 3).
Jacques, Lepercq +4 more
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