Results 171 to 180 of about 3,208,043 (303)
Super-resolution expansion microscopy in plant roots. [PDF]
Gallei M +14 more
europepmc +2 more sources
In this explorative study, the abundance of circular RNA molecules in bone marrow stem cells was found to be elevated in patients with high‐risk myelodysplastic neoplasms, and to be associated with an increased risk of progression to acute myeloid leukemia.
Eileen Wedge +17 more
wiley +1 more source
Response of plant roots to mechanical environmental stimuli. [PDF]
Baiyin B +7 more
europepmc +1 more source
The worldwide allometric relationship in anatomical structures for plant roots. [PDF]
Zhang Y +5 more
europepmc +1 more source
In the present work, we have identified a transcriptional signature based on the differential expression of six genes (BCL2&MAST4, HSH2D&LAT2, METRN&PITPNM2) that would facilitate the early detection of T‐cell acute lymphoblastic leukemia (T‐ALL) patients prone to a poor treatment response and could be implemented at diagnosis, along with other risk ...
Antonio Lahera +11 more
wiley +1 more source
Shaping with water: linking moisture perception to development in plant roots. [PDF]
Dwyer WP +2 more
europepmc +1 more source
This study shows that lung adenocarcinomas exploit developmental branching morphogenesis to acquire a therapy resistant basal‐like tumour cell state. This process was found to be regulated by combined TP53 loss‐of‐function and type‐I interferon signalling, identifying a novel axis for biomarker and therapeutic target discovery.
Kamila J Bienkowska +13 more
wiley +1 more source
Plant Roots and Phenology Drive the Spatio-Temporal Variability of Boreal Forest Floor Respiration. [PDF]
Zhou Q, Wang Z, Chen M.
europepmc +1 more source
Editorial: Increasing crop yield: the interaction of crop plant roots with their environment. [PDF]
Tracy S, Schneider H, Kant J.
europepmc +1 more source
Loss of IGF‐1R impairs DNA‐PKcs recruitment to chromatin leading to defective end‐joining
IGF‐1R promotes radioresistance by facilitating DNA‐PKcs recruitment to chromatin, enabling non‐homologous end‐joining (NHEJ) repair of double‐strand breaks. Inhibition or loss of IGF‐1R disrupts this recruitment to damage sites, driving compensatory reliance on microhomology‐mediated end‐joining (MMEJ) repair.
Matthew O. Ellis +3 more
wiley +1 more source

