Results 131 to 140 of about 12,801 (184)

Exotoxin in malaria infection (Plasmodium berghei)

Experientia, 1967
Swiss Mause, die unbehandelt eine Infektion mitPl. berghei niemals uberleben, wurden mit Serum von frisch infizierten Tieren (12. Infektionstag) immunisiert und anschliessend infiziert. Obgleich sich danach in allen Fallen eine schwere Parasitamie entwickelte, heilten 44% derjenigen Tiere nach dem 35.
S, Jerusalem, D, Bruchhausen
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Plasmodium berghei: Oxidant defense system

Experimental Parasitology, 1985
Glutathione oxidant defense system protects the erythrocyte from oxidative damage. This defense system was studied in mouse erythrocytes infected with Plasmodium berghei and in isolated parasites. The efficiency of this system was found to be increased in parasitized erythrocytes compared to the normal erythrocytes.
R K, Seth, A S, Saini, T S, Jaswal
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Characterization of Plasmodium berghei glutathione synthetase

Parasitology International, 2011
Plasmodium berghei contained 0.454±0.031 U/mg of glutathione synthetyase (GS). GS was purified using solid ammonium sulfate and Sephadex G-200 from P. berghei infected mouse erythrocytes. SDS-PAGE showed purified GS as a single band protein of 70 kDa and its Km for γ-glutamylcysteine, glycine and ATP being 0.33 mM, 8.3 mM and 0.43 mM respectively with ...
S K, Sharma, H S, Banyal
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Plasmodium berghei: The in Vitro immune response

Experimental Parasitology, 1981
Abstract Following primary in vitro Stimulation by Plasmodium berghei, IgM titers by the indirect fluorescent antibody test (IFAT) were negative in in vitro reconstituted syngeneic mouse spleen cultures containing T cells and macrophages, or B cells and macrophages, or macrophages alone, but IgM titers of 1:20 were obtained from cultures containing B
S W, Norby, N E, Alger
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Adoptive Transfer of Immunity to Plasmodium berghei

The Journal of Protozoology, 1969
SYNOPSIS. Immunity to P. berghei in rats was transferred adoptively with spleen cells but not with bone marrow cells, thymus cells, peripheral lymph node cells or thoracic duct lymphocytes from immune donors. The parasite multiplies at the same rate in control and protected rats but when about 10% of host red cells are infected the number of infected ...
J A, Roberts, P, Tracey-Patte
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Motility of Plasmodium berghei ookinetes in vitro

Journal of Invertebrate Pathology, 1975
Abstract Motility of Plasmodium berghei ookinetes, which developed in primary and established cell line cultures obtained from Anopheles stephensi mosquitoes, was studied by using still photomicrographs and normal speed cinephotomicrography. At 18–72 hr after inoculation of P.
C A, Speer, M C, Rosales-Ronquillo, P H, Silverman   +2 more
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Pre-erythrocytic Development of Plasmodium berghei

Nature, 1965
THE fate of the sporozoites of Plasmodium berghei in rodent hosts has been the subject of much research and speculation and has drawn the attention of malariologists from, the early days of the discovery of this parasite1. For it was evident to all that this useful and easily accessible Plasmodium, an instrument of great promise in biological and ...
M, YOELI, H, MOST
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In vitro Development of Plasmodium berghei Ookinetes

Nature, 1968
AFTER a mosquito has fed on an animal infected with malaria, the microgametocyte exflagellates in the mosquito gut and fertilizes the macrogamete thereby forming an ookinete. Only a few workers have studied the in vitro cultivation of the mosquito stages of malaria.
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Survival of Plasmodium berghei in Dead Hosts

The Journal of Parasitology, 1985
the adult bopyrid. The only hypothesis that seems reasonable is that sex is genetically determined in the cryptonisci of P. expansus. However, after examination of over 400 Epipenaeon ingens Nobili, only 1 juvenile female bopyrid was removed from an adult E. ingens. On morphological grounds, this juvenile proved to be P.
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Auto-Antibody in Rats with Plasmodium berghei

Nature, 1960
IN human malaria it has long been recognized that total loss of blood is significantly more extensive than loss which can be attributed to the direct rupture of parasites emerging from infected erythrocytes1. In a comparative survey of loss of blood and replacement in plasmodial infections of other mammals and birds, excessive erythrocyte destruction ...
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