Profilin reduces aggregation and phase separation of huntingtin N-terminal fragments by preferentially binding to soluble monomers and oligomers [PDF]
Aimin Li +6 more
core +2 more sources
Pcaf Modulates Polyglutamine Pathology in a <i>Drosophila</i> Model of Huntington’s Disease [PDF]
László Bodai +3 more
openalex +1 more source
Cross-Seeding Fibrillation of Q/N-Rich Proteins Offers New Pathomechanism of Polyglutamine Diseases [PDF]
Yoshiaki Furukawa +4 more
openalex +1 more source
A022 Karyopherin subunit beta 1 modulates polyglutamine-expanded huntingtin levels and fragmentation in cell models of Huntington’s disease [PDF]
N Schwarz +3 more
openalex +1 more source
Pathogenic Mechanisms of a Polyglutamine-mediated Neurodegenerative Disease, Spinocerebellar Ataxia Type 1 [PDF]
Huda Y. Zoghbi, Harry T. Orr
openalex +1 more source
A020 Characterization of protein aggregates in cell models of polyglutamine diseases [PDF]
Florian Fath +5 more
openalex +1 more source
Developing the yeast disaggregase Hsp104 as a treatment for polyglutamine disease in Drosophila
Mary Cushman Nick
openalex +1 more source
Dysregulation of gene expression in the R6/2 model of polyglutamine disease: parallel changes in muscle and brain [PDF]
Ruth Luthi‐Carter
openalex +1 more source

