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The description of molecular studies analyzing clinically diagnosed BK polyomavirus nephropathy.
, 2023 The description of molecular studies analyzing clinically diagnosed BK polyomavirus nephropathy.
Motoko Yanagita (134965) +8 more
core +1 more source
Human Polyomavirus BK Genome Analysis in BKPyV Induced Rodent Cell Lines
MicrobiologyOpen, Volume 14, Issue 5, October 2025.In this study, we analyzed the BK polyomavirus (BKPyV) genome derived from three rodent cell lines established from BKPyV induced tumors. BKPyV derived one cell line had a large deletion. The noncoding control region (NCCR) of BKPyV derived from these cell lines had the same structure as viruses induced tumors.
Setsuko Shioda +6 more
wiley +1 more source
Long‐Term Outcomes of BK Viremia in Kidney Transplant Recipients
Clinical Transplantation, Volume 39, Issue 10, October 2025.ABSTRACT Background BK viremia is associated with worse kidney transplant function and de novo donor‐specific antibodies (DSA), but mortality and graft survival are not impacted in the short‐ and intermediate‐term. The long‐term impact of BK viremia remains unclear.
Srijan Tandukar +11 more
wiley +1 more source
BKV Agnoprotein Interacts with α-Soluble N-Ethylmaleimide-Sensitive Fusion Attachment Protein, and Negatively Influences Transport of VSVG-EGFP [PDF]
, 2011 Background: The human polyomavirus BK (BKV) infects humans worldwide and establishes a persistent infection in the kidney. The BK virus genome encodes three regulatory proteins, large and small tumor-antigen and the agnoprotein, as well as the capsid ...
Johannessen, M. +23 more
core +1 more source
Clinical, genetic and molecular aspects of membranous nephropathy [PDF]
, 2011 Membranous Nephropathy (MN) is one of the leading causes of end-stage renal disease (ESRD). MN is an autoimmune disease in which autoantibodies target antigens at the level of the glomerular basement membrane.
Stanescu, H.C.
core
Polyomavirus-associated Nephropathy after Renal Transplantation [PDF]
, 2006 The first clinical infections with polyomavirus (PV) were demonstrated in 1971, when BK virus was isolated from the urine after a kidney transplant recipient and JC virus from the brain of a patient who died of progressive multifocal leukoencephalopathy.
김유선
core
Organ Medicine, Volume 2, Issue 4, Page 173-174, December 2025.
Junqi Zhang, Min Gu, Zijie Wang
wiley +1 more source
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy [PDF]
, 2011 Transforming Growth Factor-beta (TGF-β) is a pro-sclerotic cytokine widely associated with the development of fibrosis in diabetic nephropathy. Central to the underlying pathology of tubulointerstitial fibrosis is epithelial-to-mesenchymal transition ...
Hills, Claire E. +3 more
core +1 more source
Journal of the Formosan Medical Association, 2016 Polyomavirus BK (BKV) reactivation causes allograft dysfunction in some kidney transplant recipients. The use of mammalian target of rapamycin (mTOR) inhibitor-based immunotherapy is associated with a lower incidence of polyomavirus-associated ...
Chieh-Li Yen +12 more
doaj +1 more source
Molecular Networks Involved in the Immune Control of BK Polyomavirus
Clinical and Developmental Immunology, 2012 BK polyomavirus infection is the important cause of virus-related nephropathy following kidney transplantation. BK virus reactivates in 30%–80% of kidney transplant recipients resulting in BK virus-related nephropathy in 1%–10% of cases.
Eva Girmanova +6 more
doaj +1 more source