Results 161 to 170 of about 59,040 (314)
ABSTRACT Effective monitoring of aquatic biodiversity is critical for conservation, yet current approaches such as mitochondrial COI barcoding and microsatellite markers exhibit limitations in resolution, sensitivity, and scalability, particularly for detecting low‐abundance or degraded DNA in mixed aquatic samples.
Baolong Zhang +14 more
wiley +1 more source
Used version of WhatsHap for "Haplotype Threading: Accurate Polyploid Phasing from Long Reads"
Sven Schrinner +4 more
openalex +1 more source
Phylogenetic patterns and polyploid evolution within the Mediterranean genusCentaurium(Gentianaceae ‐ Chironieae) [PDF]
Guilhem Mansion +2 more
openalex +1 more source
Mitochondrial DNA Variation in the Aging Human Cerebral Cortex and Cerebellum
We used cerebellum and frontal cortex whole genome sequencing data from the North American Brain Expression Consortium to investigate mitochondrial DNA deletions, single nucleotide variants, and copy number. We found significant differences between brain regions and age effects for all these metrics, which illustrate mitochondrial changes in healthy ...
Audrey A. Omidsalar +13 more
wiley +1 more source
ABSTRACT Herbicides are a valuable tool in agricultural ecosystems to manage nuisance species. Due to the reliance on herbicides for weed control, herbicide resistance is a growing concern. Herbicides are also used extensively in aquatic and natural systems, but the genetics and evolutionary dynamics of resistance are not as frequently incorporated ...
Del Hannay +12 more
wiley +1 more source
Polyploidization delay in rat hepatocytes under liver growth inhibition by hypokinesia [PDF]
A study of young rats, weighing 55 to 59 g, after being for 10 days in conditions of limited mobility, shows a retardation of body growth as well as that of liver growth.
Brodskiy, V. Y. +3 more
core +1 more source
Development of
Juan Ling +13 more
openalex +1 more source
ABSTRACT Glanzmann thrombasthenia (GT) is an inherited hemorrhagic disorder characterised by impaired platelet functions, manifested clinically as spontaneous bleeding. It is usually inherited in an autosomal recessive manner. Platelet dysfunction in patients with GT is caused by quantitative and/or qualitative deficiencies in αIIbβ3, which result from
Jiao Wu +14 more
wiley +1 more source

