Accidental Organophosphate Poisoning in a Patient Due to a Bed Bug Pesticide in the United Kingdom: A Case Report. [PDF]
Griffiths D +4 more
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Neuroprotective Effects of N-Acetylcysteine-Amide (AD4) in a Survival Mouse Model of Paraoxon Intoxication: Targeting Oxidative Stress, Neuroinflammation and Memory Impairments. [PDF]
Urquizu E +7 more
europepmc +1 more source
Microbiological Etiology and Detection of Drug-Resistant Bacteria in Pneumonia in Patients with Organophosphate Poisoning in the Intensive Care Unit. [PDF]
Kanno N +16 more
europepmc +1 more source
Analysis of risk factors for severe acute poisoning in children. [PDF]
Li J, Wang WK.
europepmc +1 more source
Determinants of Poor Outcomes in Acute Poisoning at a Tertiary Care Hospital in an Industrial City of Eastern India: A Retrospective Observational Study. [PDF]
Khan MA, Pathak S, Khan MKJ, Mishra N.
europepmc +1 more source
Protective effects of lipid emulsion on vital organs through the LPS/TLR4 pathway in acute organophosphate poisoning. [PDF]
Li G, Hu H.
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Therapeutic Dosing of Pralidoxime Chloride
Pralidoxime chloride is a useful agent in the treatment of organophosphate poisoning. Poisindex, a widely used poisoning treatment resource, recommends dosing pralidoxime chloride as an intermittent iv infusion every 8–12 hours, whereas other authors have used continuous iv infusion with good results.
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Pralidoxime in carbaryl poisoning: an animal model
Human and Experimental Toxicology, 2007Introduction: Poisoning from organophosphates and carbamates is a significant cause of morbidity and mortality worldwide. Concerns have been expressed over the safety and efficacy of the use of oximes such as pralidoxime (2-PAM) in patients with carbamate poisoning in general, and more so with carbaryl poisoning specifically.
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