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Pathophysiology of premature aging characteristics in Mendelian progeroid disorders.
European Journal of Medical Genetics, 2020Aging is a physiological process that is in part genetically determined. Some of the signs and symptoms of aging also occur prematurely in Mendelian disorders.
R. Hennekam
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Premature Birth and Age at Onset of Puberty
Epidemiology, 2012Premature birth is associated with poor metabolic health in both sexes, potentially via earlier pubertal timing.We examined the associations of gestational age and premature birth (< 37 weeks gestation) with age at onset of puberty (Tanner stage II for breast or genitalia development).
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Growth Hormone and Premature Ageing
Nature, 1973IT has often been suggested that the immune system may be involved in ageing1–4 Fabris et al.5 claim that such immune deficiencies are caused by inadequate stimulation of the lymphoid system by growth hormone (GH). They compared mice with inherited pituitary dwarfism (homozygotes for Snell's dwarf mutation, dw) with normal mice from the same stock. The
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Searching for clues to premature aging
Trends in Endocrinology & Metabolism, 2002Hutchinson-Gilford Progeria syndrome is a rare developmental disorder affecting most of the organ systems in a manner that mimics natural aging but at a markedly accelerated rate. A recent workshop at the NIH, aimed at further understanding the aging process, took an in-depth look at this complex syndrome.
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Nature Medicine, 2008
Two commonly prescribed drugs, statins and aminobisphosphonates, may be helpful in combating the rare aging disorder, Hutchinson-Gilford progeria syndrome (pages 767–772).
Eran Meshorer, Yosef Gruenbaum
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Two commonly prescribed drugs, statins and aminobisphosphonates, may be helpful in combating the rare aging disorder, Hutchinson-Gilford progeria syndrome (pages 767–772).
Eran Meshorer, Yosef Gruenbaum
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2012
Cockayne Syndrome – De Barsy Syndrome – Hallermann-Streiff Syndrome – Hutchinson-Gilford Progeria – Werner ...
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Cockayne Syndrome – De Barsy Syndrome – Hallermann-Streiff Syndrome – Hutchinson-Gilford Progeria – Werner ...
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Science, 2018
Premature Aging Cells from patients with Hutchinson-Gilford progeria syndrome (HGPS) have defects in nuclear architecture that lead to premature cellular senescence. Larrieu et al. investigated the mechanisms by which a small molecule called remodelin improves the phenotype of HGPS cells (see the Focus by Wilson).
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Premature Aging Cells from patients with Hutchinson-Gilford progeria syndrome (HGPS) have defects in nuclear architecture that lead to premature cellular senescence. Larrieu et al. investigated the mechanisms by which a small molecule called remodelin improves the phenotype of HGPS cells (see the Focus by Wilson).
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Current Opinion in Infectious Diseases, 2012
Martin, Fisher, Vanessa, Cooper
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Martin, Fisher, Vanessa, Cooper
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