Results 81 to 90 of about 23,181 (145)

Loss of SMARCB1 evokes targetable epigenetic vulnerabilities in epithelioid sarcoma

open access: yes
Cancer Communications, Volume 45, Issue 5, Page 494-499, May 2025.
Jia Xiang Jin   +12 more
wiley   +1 more source

Understanding Functional Protein-Protein Interactions Of ABCB11 And ADA In Human And Mouse [PDF]

open access: yesarXiv, 2015
Proteins are macromolecules which hardly act alone; they need to make interactions with some other proteins to do so. Numerous factors are there which can regulate the interactions between proteins [4]. Here in this present study we aim to understand Protein -Protein Interactions (PPIs) of two proteins ABCB11 and ADA from quantitative point of view ...
arxiv  

HSEARCH: fast and accurate protein sequence motif search and clustering [PDF]

open access: yesarXiv, 2017
Protein motifs are conserved fragments occurred frequently in protein sequences. They have significant functions, such as active site of an enzyme. Search and clustering protein sequence motifs are computational intensive. Most existing methods are not fast enough to analyze large data sets for motif finding or achieve low accuracy for motif clustering.
arxiv  

Synovial fibroblasts: key players in rheumatoid arthritis [PDF]

open access: yes, 2017
Rheumatoid arthritis (RA) is a chronic autoimmune-disease of unknown origin that primarily affects the joints and ultimately leads to their destruction. The involvement of immune cells is a general hallmark of autoimmune-related disorders. In this regard,
Distler, O.   +5 more
core  

Protein Hypernetworks: a Logic Framework for Interaction Dependencies and Perturbation Effects in Protein Networks [PDF]

open access: yesarXiv, 2011
Motivation: Protein interactions are fundamental building blocks of biochemical reaction systems underlying cellular functions. The complexity and functionality of such systems emerge not from the protein interactions themselves but from the dependencies between these interactions.
arxiv  

Proto-oncogene c-jun expression is induced by AML1-ETO in a JNK dependent manner:possible role in the pathogenesis of acute myeloid leukemia [PDF]

open access: yes, 2003
Overexpression of proto-oncogene c-jun and constitutive activation of the Jun NH2-terminal kinase (JNK) signaling pathway have been implicated in the leukemic transformation process.
Elsaesser, Annika
core  

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