Results 211 to 220 of about 201,089 (265)
Systemic aging fuels heart failure: Molecular mechanisms and therapeutic avenues
ESC Heart Failure, Volume 12, Issue 2, Page 1059-1080, April 2025.Abstract Systemic aging influences various physiological processes and contributes to structural and functional decline in cardiac tissue. These alterations include an increased incidence of left ventricular hypertrophy, a decline in left ventricular diastolic function, left atrial dilation, atrial fibrillation, myocardial fibrosis and cardiac ...
Zhuyubing Fang +7 more
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Biomarkers of lung congestion and injury in acute heart failure
ESC Heart Failure, Volume 12, Issue 2, Page 781-789, April 2025.Abstract Acute heart failure (AHF) classification and management are primarily based on lung congestion and/or hypoperfusion. The quantification of the vascular and tissue lung damage is not standard practice though biomarkers of lung injury may play a relevant role in this context.
Marco Guazzi +9 more
wiley +1 more source
ESC Heart Failure, Volume 12, Issue 2, Page 942-954, April 2025.Abstract The immune system has long been recognized as a key driver in the progression of heart failure (HF). However, clinical trials targeting immune effectors have consistently failed to improve patient outcome across different HF aetiologies. The activation of the immune system in HF is complex, involving a broad network of pro‐inflammatory and ...
Johann Roessler +4 more
wiley +1 more source
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European Radiology, 1998
Evaluation of the pulmonary vasculature is mainly indicated in patients with suspected pulmonary thromboembolism. The routine procedure so far is ventilation-perfusion scintigraphy alone or in combination with diagnostic assessment of the legs to rule out deep venous thrombosis. The results are still not reliable for the majority of patients.
Bongartz, G. +3 more
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Evaluation of the pulmonary vasculature is mainly indicated in patients with suspected pulmonary thromboembolism. The routine procedure so far is ventilation-perfusion scintigraphy alone or in combination with diagnostic assessment of the legs to rule out deep venous thrombosis. The results are still not reliable for the majority of patients.
Bongartz, G. +3 more
openaire +3 more sources
Pulmonary Circulation at Exercise
Comprehensive Physiology, 2012Abstract The pulmonary circulation is a high‐flow and low‐pressure circuit, with an average resistance of 1 mmHg/min/L in young adults, increasing to 2.5 mmHg/min/L over four to six decades of life. Pulmonary vascular mechanics at exercise are best described by distensible models.
Naeije, Robert, Chesler, Naomi C
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STUDIES OF THE PULMONARY CIRCULATION [PDF]
Journal of Experimental Medicine, 1911 exaly +3 more sources
JAMA: The Journal of the American Medical Association, 1978
THE HISTORY of the pulmonary circulation provides a measure of man's thinking about himself and his place in the universe. For centuries before De Motu Cordis in 1628, 1 the blood vessels of the lungs were incorporated into an elaborate scheme that explained the origin and distribution of spirits that animated and sustained the human being.
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THE HISTORY of the pulmonary circulation provides a measure of man's thinking about himself and his place in the universe. For centuries before De Motu Cordis in 1628, 1 the blood vessels of the lungs were incorporated into an elaborate scheme that explained the origin and distribution of spirits that animated and sustained the human being.
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Pharmacology of the Pulmonary Circulation
2011The pulmonary vasculature is a complex system, and studies of the effects of anesthetic drugs on this system are often contradictory. A balanced anesthetic technique with adherence to the hemodynamic goals of maintenance of right ventricular preload and right coronary artery perfusion is the safest choice for patients with PHTN.
Cara Reimer, John Granton
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Digitalis and the pulmonary circulation
American Heart Journal, 1961Abstract In anesthetized dogs, the intravenous injection of acetyl strophanthidin caused a slight decrease in pulmonary arterial pressure which could not be explained by the effect on left atrial pressure. There was a marked decrease in the pulmonary venous flow, which was often accompanied by cardiac slowing and an increase in the calculated ...
Y S, KIM, D M, AVIADO
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