Results 201 to 210 of about 261,257 (360)
ABSTRACT Background Prurigo nodularis patients face a significant burden of comorbidities, necessitating a comprehensive approach to care. We aimed to explore the association between prurigo nodularis, its comorbid conditions, and healthcare usage patterns.
Yuliya Valdman‐Grinshpoun +4 more
wiley +1 more source
Data-driven cluster analysis and external validation identify phenotypic subgroups in renin-independent aldosteronism with differential cardiovascular risk and therapeutic implications. [PDF]
Liu Y +11 more
europepmc +1 more source
Total serum T3 and T4 in nontoxic goiter and autonomous adenoma [PDF]
Borowzak, H. +5 more
core +1 more source
This study focuses on the disease burden of chronic kidney disease due to type 2 diabetes (CKD due to type 2 diabetes) globally and in China from 1990 to 2021 and predicts its trend from 2022 to 2031. ABSTRACT Objective To analyze the disease burden of chronic kidney disease due to diabetes mellitus type 2 (CKD due to diabetes mellitus type 2) in China
Jinpeng Du +5 more
wiley +1 more source
Hormonal response following hemorrhage after severe trauma: an observational prospective study. [PDF]
Werner M +9 more
europepmc +1 more source
ABSTRACT Background Inflammation plays an essential role in the pathogenesis of diabetic nephropathy (DN). Linderalactone (LNL), as a natural sesquiterpene lactone, has been discovered to have anti‐inflammatory activation. However, the effects of linderalactone on diabetes‐associated renal damage remain unclear.
Hao Chen +7 more
wiley +1 more source
Subclinical primary aldosteronism. [PDF]
Parisien-La Salle S +4 more
europepmc +1 more source
Renal renin-angiotensin system —What supports and breaks the circle of life—
Shoji Kagami
openalex +2 more sources
The biology of PKM2 in the metabolism and senescence in diabetic kidney disease
ABSTRACT Diabetic kidney disease (DKD) is one of the most common causes of chronic kidney disease that leads to end‐stage kidney disease, and its progression is closely linked to metabolic stress within renal tubular cells. Under long‐term hyperglycemia, cells shift their glucose metabolism from normal oxidative phosphorylation toward glycolysis.
Md. Imrul Kayes, Keizo Kanasaki
wiley +1 more source

