Results 171 to 180 of about 267,492 (314)

Mechanisms underlying leptininduced cardioprotection

open access: yes, 2010
BACKGROUND: The tissue damage resulting from myocardial ischaemia/reperfusion (I/R) leads to a large proportion of the deaths in the developed world.
Dixon, R.A.
core  

Possible therapeutic repositioning of valproic acid: From epileptic seizures to acute kidney injury

open access: yesBritish Journal of Clinical Pharmacology, EarlyView.
Valproic acid, an anticonvulsant, may be repositioned to prevent acute kidney injury due to ischemia followed by reperfusion. It preserves renal functions, electrolyte homeostasis and active sodium transport in kidney tubules, and blocks the onset of hypertension.
Danilo Alves‐Bezerra   +8 more
wiley   +1 more source

Steric modulation of nitric oxide photodissociation kinetics in iron–nitrosyl complexes

open access: yesBulletin of the Korean Chemical Society, EarlyView.
Subtle steric modification within a pyridinophane ligand framework modulates nitric oxide photodissociation kinetics in iron–nitrosyl complexes. Structural and electrochemical analyses reveal that modest ligand‐environment variations are reflected in differences in NO photoreactivity.
Jisu Choe, Jaeheung Cho
wiley   +1 more source

Deciphering the catalytic and pharmacological mechanisms of Coptis chinensis herbzymes to renovate intestinal microenvironment for colitis alleviation

open access: yesBMEMat, EarlyView.
The synthesized CCzymes possess both antioxidant enzyme activity and pharmacological properties inherent to Coptis chinensis. By their antioxidant enzyme activity, CCzymes can attenuate oxidative stress within the inflammatory region of ulcerative colitis (UC), while their pharmacological activity acts on macrophage polarization and the intestinal ...
Zhichao Deng   +10 more
wiley   +1 more source

Cardioprotective Effect of Aloin Against Ischemia/Reperfusion Injury: Experimental and Computational POM Approaches. [PDF]

open access: yesJ Biochem Mol Toxicol
Kadi M   +12 more
europepmc   +1 more source

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