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Pulmonary Reperfusion Syndrome
The Annals of Thoracic Surgery, 1979"Reperfusion syndrome" of the lung may play a role in the pulmonary edema and hemorrhage that occur following pulmonary embolectomy, cardiopulmonary bypass, and shock. Bioenergetic, metabolic, and ultrastructural studies of canine lungs indicate that ventilated lung tissue could tolerate 5 hours of pulmonary arterial occlusion with minimal damage ...
D L, Modry, R C, Chiu
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Controlled Reperfusion Against Ischemia Reperfusion Injury
2018Recanalization and reperfusion after an ischemic stroke sometimes could be harmful and deadly, and controlled perfusion may be a potential solution. This article summarized controlled perfusion literature, both animal and clinical, on heart, lung, kidney, and limbs and compared with brain controlled perfusion.
Weijian Jiang +3 more
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Critical Care Nursing Clinics of North America, 1990
A review of the mechanism of ischemic-reperfusion injury, proposed interventions to prevent injury, and future directions have been presented to enhance the practitioner's knowledge of this new, exciting concept in myocardial injury. There is increasing evidence in the literature that reperfusion injury may occur in other organ systems and is ...
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A review of the mechanism of ischemic-reperfusion injury, proposed interventions to prevent injury, and future directions have been presented to enhance the practitioner's knowledge of this new, exciting concept in myocardial injury. There is increasing evidence in the literature that reperfusion injury may occur in other organ systems and is ...
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New England Journal of Medicine, 2007
The pathogenesis of an acute myocardial infarction consists of the rupture of atherosclerotic plaque, followed by a sudden thrombotic coronary occlusion.
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The pathogenesis of an acute myocardial infarction consists of the rupture of atherosclerotic plaque, followed by a sudden thrombotic coronary occlusion.
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Controlled perfusion decreases reperfusion injury after high-flow reperfusion
The Journal of Heart and Lung Transplantation, 2002Some investigators have suggested that high pulmonary artery flow rates increase the risk of severe reperfusion injury after lung transplantation. We hypothesized that controlling the initial flow rate and pulmonary artery pressure would decrease the severity of lung dysfunction in the setting of high-flow reperfusion.Using our isolated, ventilated ...
Steven M, Fiser +7 more
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Hyperoxia during reperfusion is a factor in reperfusion injury
Free Radical Biology and Medicine, 1989Imposition of ischemia should result in accumulation of lactic acid with an attendant drop in pH. Subsequent reperfusion would result in hyperoxia, in the affected tissue, due to the Bohr Effect. O2- should therefore be produced in greater than normal amounts, due to this transient hyperoxia, and may contribute to reperfusion injury.
M L, Wolbarsht, I, Fridovich
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Seminars in Cardiothoracic and Vascular Anesthesia, 2006
Cycles of ischemia-reperfusion are ubiquitous in clinical cardiology. Depending on the duration and intensity of the ischemic episode as well as its repetition mode, several pathophysiologic syndromes have been identified, such as myocardial stunning, hibernation, and preconditioning.
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Cycles of ischemia-reperfusion are ubiquitous in clinical cardiology. Depending on the duration and intensity of the ischemic episode as well as its repetition mode, several pathophysiologic syndromes have been identified, such as myocardial stunning, hibernation, and preconditioning.
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Reperfused Myocardium: Stunning, Preconditioning, and Reperfusion Injury
1990In the late 1960s, when the deathtoll from myocardial infarction had caused a high level of public awareness, the National Institutes of Health, USA, decided to fund projects directly related to the experimental treatment of infarcts, to develop animal models best suited to study infarct size after coronary occlusion, and to develop quantitative ...
Wolfgang Schaper +2 more
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1989
Thrombolysis with intravenous streptokinase, APSAC and tissue plasminogen activator has been shown to improve ventricular function and survival in patients with acute myocardial infarction (1–4). Despite these important advances several problems remain and we may not have achieved the optimal benefit from reperfusion therapy.
B. Pitt, B. Lucchesi
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Thrombolysis with intravenous streptokinase, APSAC and tissue plasminogen activator has been shown to improve ventricular function and survival in patients with acute myocardial infarction (1–4). Despite these important advances several problems remain and we may not have achieved the optimal benefit from reperfusion therapy.
B. Pitt, B. Lucchesi
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