Results 171 to 180 of about 91,701 (297)
A nonpigmented retinal pigment epithelium adenoma with retinitis pigmentosa:A case report. [PDF]
Zhu X, Zhu Y, Yang X, Liu H.
europepmc +1 more source
The prechoroidal cleft in neovascular age‐related macular degeneration
Abstract The prechoroidal cleft is a lenticular, hypo‐reflective space on optical coherence tomography imaging, located between a band of fibrovascular material underneath the retinal pigment epithelium (RPE) and Bruch's membrane. It occurs in 8%–22% of neovascular age‐related macular degeneration (nAMD) eyes, most often with macular neovascularization
Niels J. Brouwer +3 more
wiley +1 more source
AGE-RELATED RETINAL PIGMENT EPITHELIUM DISEASE: Clinical and Differential Features. [PDF]
Arrigo A +7 more
europepmc +1 more source
Abstract Central serous chorioretinopathy (CSC) is a chorioretinal disease characterised by serous subretinal fluid (SRF) in the macula, resulting in sudden central vision loss. It predominantly affects working‐age adults, particularly men aged 30 to 60 years.
I. Made Ferdiko Hutamadella +10 more
wiley +1 more source
Retinal pigment epithelium pseudopods reach across the divide to maintain photoreceptor performance and save vision. [PDF]
Sodhi A.
europepmc +1 more source
Ca2+‐Activated Cl− Channels: Do Bestrophins and TMEM16A Interact?
ABSTRACT Aim Ca2+‐activated Cl– conductances are present in many cell types and are important for regulating membrane potential as well as other cellular functions. TMEM16A is widely accepted as the principal molecular basis for Ca2+‐activated Cl– conductances, but also members of the bestrophin family may be important for some Ca2+‐activated Cl ...
Christian Aalkjær +4 more
wiley +1 more source
A case of congenital simple hamartoma of the retinal pigment epithelium combined with fascicular nerve fibre layer defects. [PDF]
Duan HT, Dong M, Kang L.
europepmc +1 more source
GRKs and arrestins: Nomenclature and functions in GPCR‐dependent and ‐independent signalling
G protein‐coupled receptor (GPCR) kinases (GRKs) and arrestins play a critical role in the regulation of GPCR signalling. Historic names of mammalian GRKs were replaced by systematic ones in the 1990s; however, both kinds of names are currently in use for mammalian arrestins.
Vsevolod V. Gurevich
wiley +1 more source

