Results 181 to 190 of about 104,378 (298)

Decoding the Role of Caveolin‐1 in Morphological Diversity and Self‐Renewal of Breast Cancer Cells

open access: yesCell Proliferation, EarlyView.
The silence of caveolin‐1 (Cav‐1) reproduced the morphological evolutionary behaviour of cancer cells, which is similar to the epithelial‐mesenchymal transition process. And Cav‐1 dependent morphological changes could affect cancer cell self‐renewal capacity through maintaining the morphological stability.
Shun Li   +14 more
wiley   +1 more source

Endothelial Nucleoporin93 (Nup93) Maintains Vascular Function via Sun1-Dependent Regulation of RhoA-eNOS Signaling [PDF]

open access: green
Tung Nguyen   +10 more
openalex   +1 more source

Enitociclib (VIP152), venetoclax and prednisone in relapsed or refractory aggressive non‐Hodgkin lymphoma

open access: yes
British Journal of Haematology, EarlyView.
Max J. Gordon   +12 more
wiley   +1 more source

Primary Cutaneous Peripheral T‐Cell Lymphoma With Follicular Helper T‐Cell Phenotype

open access: yesJournal of Cutaneous Pathology, EarlyView.
ABSTRACT An otherwise healthy 82‐year‐old woman presented with recurrent, asymptomatic erythematous papulo‐nodular lesions on the face and extremities. A skin biopsy revealed a dense, non‐epidermotropic lymphohistiocytic infiltrate with granulomatous features and a mixed population of medium‐sized atypical lymphoid cells and plasma cells ...
Nidia Planella‐Fontanillas   +9 more
wiley   +1 more source

Kinesin-1 coordinates cross-talk between microtubule and actin cytoskeletons during dendritic cell migration. [PDF]

open access: yesSci Adv
Duquesne P   +19 more
europepmc   +1 more source

EphB-Mediated Degradation of the RhoA GEF Ephexin5 Relieves a Developmental Brake on Excitatory Synapse Formation [PDF]

open access: bronze, 2010
Seth S. Margolis   +12 more
openalex   +1 more source

Combating muscle atrophy: emerging therapeutic targets that are fiber‐type‐specific

open access: yesThe FEBS Journal, EarlyView.
Inflammation, denervation or decreased insulin signaling activate proteolysis and cause muscle wasting. Pathological conditions like obesity and DMD can also exacerbate atrophy via PGD2–RhoA–ROCK2 signaling. In contrast, exercise, dietary interventions, and pharmacological agents can activate Ca2+/calcineurin–NFAT and AMPK–PGC‐1α–MEF pathways ...
Samrat Chakraborty   +2 more
wiley   +1 more source

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