Results 91 to 100 of about 9,101,925 (291)

Why Are African Researchers Left Behind in Global Scientific Publications? – A Viewpoint [PDF]

International Journal of Health Policy and Management
Why Are African Researchers Left Behind in Global Scientific Publications?
Juliet Nabyonga-Orem, James Avoka Asamani, Olushayo Olu   +2 more
doaj   +1 more source

CEACAM1 participation in breast cancer progression

Molecular Oncology, EarlyView.
In invasive breast cancer (BC), CEACAM1 shifts from an apical to a uniform membranous/cytoplasmic pattern, or is lost, as tumors dedifferentiate, inversely tracking the Ki‐67 proliferative index. In MCF‐7 cells, only CEACAM1‐4L suppresses proliferation, repressing cell cycle and growth factor genes.
Mykola Lyndin, Irina Kube‐Golovin, Anatolii Romaniuk, Gunther Wennemuth   +3 more
wiley   +1 more source

Victoria Falls green map

, 2001
A map of Victoria Falls, Zimbabwe, featuring the area's flora, fauna, environmental concerns as well as sites of culture and ...
Victoria Falls Environmental Capacity and Master Plan Project
core   +1 more source

Building Capacity For Innovation Through R&D Consortia in Health Projects: From Network Interaction to Systemic Transformation

STIPM (STI Policy and Management) Journal, 2017
This paper attempt to elucidate the postulation that there is a continuous trajectory from the network interaction in scientific research towards the systemic transformation in innovation capacity building. This study will expose that the availability of
Ria Hardiyati, Trina Fizzanty, Erman Aminullah   +2 more
doaj   +1 more source

Extracellular vesicle lipidomics liquid biopsy reveals systemic and tumor microenvironment metabolic reprogramming in ovarian cancer

Molecular Oncology, EarlyView.
Extracellular vesicle (EV) lipidomic profiling of plasma and ascites from patients with high‐grade serous ovarian cancer and of ovarian cancer cell lines reveals enrichment of triglycerides (TGs), diacylglycerols (DGs), phosphatidylcholine (PCs), sphingomyelins (SMs), reflecting tumor metabolic reprogramming.
Shikha Rani, Dominic Guanzon, Andrew Lai, Katherin Scholz‐Romero, Sarah Reed, Edita Ritmejeryte, Rohan Lourie, Naven Chetty, Kaltin Ferguson, Carlos Salomon   +9 more
wiley   +1 more source

NAPRT loss promotes lung tumor initiation and growth through AKT signaling independently of NAD+ biosynthesis

Molecular Oncology, EarlyView.
Loss of NAPRT promotes lung tumor initiation and growth through a noncanonical mechanism, independent of its role in NAD+ biosynthesis. Mechanistically, NAPRT depletion activates the mTORC2‐driven AKT/β‐catenin signaling axis to enhance clonogenic and invasive phenotypes. Furthermore, lung‐specific Naprt deletion significantly increases tumor burden in
Myung Joon Oh, Ji Yun Jang, Yoon Jeon, Gi‐Cheon Kim, Hwanho Lee, Jooyoung Lee, Ju‐Hwa Kim, Hakhyun Kim, Minjee Kim, Ho‐Keun Kwon, Ho Lee, Hyun Seok Kim   +11 more
wiley   +1 more source

Division of Global Public Health Capacity Development 2008 annual report [PDF]

Based in Atlanta, Georgia (U.S.A.), the Division of Global Public Health Capacity Development (DGPHCD) is part of the Coordinating Office for Global Health at the U .S . Centers for Disease Control and Prevention.

core  

DELTAS CPE Seed Fund Evaluation - Data set and supplementary information

DELTAS CPE Seed Fund Evaluation - Data set and supplementary ...
Centre for Capacity Research
core   +1 more source

Study on the Influence of Academic Scientific Communities in the Capacity for Sustainable Development of College Students

SHS Web of Conferences, 2015
The rapid social development and speedy growth of the total information amount put forward ever higher demands on the sustainable capacity of college students. As a brand-new way to train college talents, academic scientific communities play a vital role
Zhang Jin
doaj   +1 more source

Regulation of the lncRNA NEAT1 by p53‐ΔNp63 crosstalk modulates the DNA damage response and therapeutic efficacy in HNSCC

Molecular Oncology, EarlyView.
In head and neck squamous cell carcinoma (HNSCC) p53 and p63 exert opposite roles on the transcription regulation of the lncRNA NEAT1. Under basal conditions, p53 levels are low and p63 represses NEAT1 expression. Upon genotoxic stress, p53 is rapidly induced, displacing p63 from the NEAT1 promoter leading to NEAT1 transcriptional activation and ...
Sara De Domenico, Veronica La Banca, Silvia D'Amico, Stefano Scalera, Sara Nicolai, Angelo Peschiaroli   +5 more
wiley   +1 more source