Results 161 to 170 of about 100,466 (204)
Some of the next articles are maybe not open access.
Survey of Anesthesiology, 1962
Abstract 1. 1. A review of the rationale for the use of hypothermia in infectious shock, especially severe peritonitis, is presented. 2. 2. Experience in the use of hypothermia in eighteen patients with severe peritonitis is presented and discussed. 3. 3. Hypothermia is an adjuvant in the therapy of infectious shock and severe peritonitis,
openaire +5 more sources
Abstract 1. 1. A review of the rationale for the use of hypothermia in infectious shock, especially severe peritonitis, is presented. 2. 2. Experience in the use of hypothermia in eighteen patients with severe peritonitis is presented and discussed. 3. 3. Hypothermia is an adjuvant in the therapy of infectious shock and severe peritonitis,
openaire +5 more sources
Septic Shock (Endotoxic Shock)
Clinical Obstetrics and Gynecology, 1973PIP: The pathophysiology, clinical aspects, medical, and surgical management of endotoxin shock are reviewed. In the primate, the pathophysiology of endotoxin shock is contributed to by selective vasopasm, disseminated intravascular coagulation, and reduced myocardial response to sympathetic stimuli. Studies in the baboon measured various parameters of
Papineni S. Rao, Denis Cavanagh
openaire +3 more sources
Critical Care Medicine, 1983
Naloxone, 0.3 mg/kg of a 10 mg/ml solution, was administered as a single bolus to patients in septic shock if their systolic blood pressure (BP) was less than 100 mm Hg or MAP less than 70 mm Hg with evidence of renal or cerebral hypoperfusion. Patients with chronic or acute (less than 12 h) administration of narcotics were excluded.
William S. Howland +2 more
openaire +2 more sources
Naloxone, 0.3 mg/kg of a 10 mg/ml solution, was administered as a single bolus to patients in septic shock if their systolic blood pressure (BP) was less than 100 mm Hg or MAP less than 70 mm Hg with evidence of renal or cerebral hypoperfusion. Patients with chronic or acute (less than 12 h) administration of narcotics were excluded.
William S. Howland +2 more
openaire +2 more sources
Obstetrics & Gynecology, 1997
To evaluate the etiology, management, and maternal and perinatal outcome in patients with septic shock during pregnancy.In 18 patients with septic shock during pregnancy, the criteria for the diagnosis were sepsis-induced hypotension unresponsive to adequate fluid resuscitation and requirement for vasopressors.Causes of shock were pyelonephritis (n = 6)
William C. Mabie +5 more
openaire +2 more sources
To evaluate the etiology, management, and maternal and perinatal outcome in patients with septic shock during pregnancy.In 18 patients with septic shock during pregnancy, the criteria for the diagnosis were sepsis-induced hypotension unresponsive to adequate fluid resuscitation and requirement for vasopressors.Causes of shock were pyelonephritis (n = 6)
William C. Mabie +5 more
openaire +2 more sources
Septic Shock and Corticosteroids
New England Journal of Medicine, 1981SCOPUS: le.j ; info:eu-repo/semantics ...
openaire +3 more sources
American Journal of Obstetrics and Gynecology, 1962
Abstract 1. 1. A series of 12 cases of septic abortion with shock due to infection are presented. These occurred in 662 cases of septic abortion seen in Detroit Receiving Hospital in the past 2 years. 2. 2. The early recognition of these cases and the institution of massive antibiotic therapy is of crucial importance, since such therapy is ...
Cheng-Chao Yang +3 more
openaire +3 more sources
Abstract 1. 1. A series of 12 cases of septic abortion with shock due to infection are presented. These occurred in 662 cases of septic abortion seen in Detroit Receiving Hospital in the past 2 years. 2. 2. The early recognition of these cases and the institution of massive antibiotic therapy is of crucial importance, since such therapy is ...
Cheng-Chao Yang +3 more
openaire +3 more sources
The haemodynamics of septic shock
Intensive Care Medicine, 1990Both peripheral vascular abnormalities and changes in myocardial function are hallmarks of septic shock. Their complex interactions result in inadequate and maldistributed microcirculatory flow and progressive organ dysfunction. Inappropriate vasodilation, microembolization and endothelial cell injury are proposed mechanisms that may induce ...
A. J. Schneider +2 more
openaire +3 more sources
Pathophysiology of Septic Shock
Critical Care Clinics, 2018Fundamental features of septic shock are vasodilation, increased permeability, hypovolemia, and ventricular dysfunction. Vasodilation owing to increased nitric oxide and prostaglandins is treated with vasopressors (norepinephrine first). Increased permeability relates to several pathways (Slit/Robo4, vascular endothelial growth factor, angiopoietin 1 ...
John H. Boyd +2 more
openaire +2 more sources
2023
The body’s response to infection consists of the release of various cytokines and mediators inducing a state of vasoplegia clinically characterized by hypotension, increased heart rate, and decreased cardiac output. The hyperdynamic low-pressure state, which we recognizes in clinical practice, is usually the consequence of the initial fluid ...
openaire +2 more sources
The body’s response to infection consists of the release of various cytokines and mediators inducing a state of vasoplegia clinically characterized by hypotension, increased heart rate, and decreased cardiac output. The hyperdynamic low-pressure state, which we recognizes in clinical practice, is usually the consequence of the initial fluid ...
openaire +2 more sources
1997
Despite significant improvements in critical care, septic shock remains the major cause of death in noncoronary intensive care units with an estimated mortality (gram-negative and gram-positive sepsis) ranging between 25% and 75%. Traditionally recognized as a consequence of gram-negative bacteremia, septic shock is also caused by gram-positive ...
Christoph Thiemermann +1 more
openaire +2 more sources
Despite significant improvements in critical care, septic shock remains the major cause of death in noncoronary intensive care units with an estimated mortality (gram-negative and gram-positive sepsis) ranging between 25% and 75%. Traditionally recognized as a consequence of gram-negative bacteremia, septic shock is also caused by gram-positive ...
Christoph Thiemermann +1 more
openaire +2 more sources