Results 41 to 50 of about 39,748 (262)

A Graph Convolution for Signed Directed Graphs

open access: yesCoRR, 2022
Preprint ...
Ko, Taewook, Kim, Chong-Kwon
openaire   +2 more sources

Salmonella lipopolysaccharide‐containing supported lipid bilayers as platforms to study bacteriophage interactions

open access: yesFEBS Letters, EarlyView.
We present robust protocols for the preparation of supported lipid bilayers (SLBs) incorporating either Salmonella smooth LPS or outer membrane vesicles (OMVs). We use a combination of quartz crystal microbalance with dissipation (QCM‐D) and fluorescence microscopy to both characterize the SLBs of various compositions and to probe their interactions ...
Hudson P. Pace   +6 more
wiley   +1 more source

Further Results on the Nullity of Signed Graphs

open access: yesJournal of Applied Mathematics, 2014
The nullity of a graph is the multiplicity of the eigenvalue zero in its spectrum. A signed graph is a graph with a sign attached to each of its edges. In this paper, we apply the coefficient theorem on the characteristic polynomial of a signed graph and
Yu Liu, Lihua You
doaj   +1 more source

On sign-invertible graphs

open access: yesDiscrete Applied Mathematics
Let $G$ be a graph and $A$ be its adjacency matrix. A graph $G$ is invertible if its adjacency matrix $A$ is invertible and the inverse of $G$ is a weighted graph with adjacency matrix $A^{-1}$. A signed graph $(G,σ)$ is a weighted graph with a special weight function $σ: E(G)\to \{-1,1\}$. A graph is sign-invertible (or sign-invertible) if its inverse
Isaiah Osborne, Dong Ye 0002
openaire   +2 more sources

From mice to humans—divergent strategies for intestinal homeostasis and regeneration

open access: yesFEBS Letters, EarlyView.
Recent advances such as organoid genome editing, xenotransplantation, imaging, and whole‐genome sequencing have enabled direct studies of human intestinal stem cells (ISCs). These studies reveal species‐specific features, including slower ISC proliferation, distinct injury responses, slower somatic mutation accumulation in humans, and an inverse ...
Keiko Ishikawa   +2 more
wiley   +1 more source

On Regular Signed Graphs with Three Eigenvalues

open access: yesDiscussiones Mathematicae Graph Theory, 2020
In this paper our focus is on regular signed graphs with exactly 3 (distinct) eigenvalues. We establish certain basic results; for example, we show that they are walk-regular.
Anđelić Milica   +2 more
doaj   +1 more source

Design and analysis strategies for robust microbiome ageing research

open access: yesFEBS Letters, EarlyView.
The gut microbiome changes with age and associates with age‐related morbidity and mortality, establishing it as a potential biomarker and intervention target for ageing. Realising this potential requires methodological rigour, yet distinguishing biological signals from methodological artefacts remains challenging across cohorts. This review provides an
Mark Olenik   +5 more
wiley   +1 more source

Star complements for ±2 in signed graphs

open access: yesSpecial Matrices, 2022
In this article, we investigate connected signed graphs which have a connected star complement for both −2-2 and 2 (i.e. simultaneously for the two eigenvalues), where −2-2 (resp.
Mulas Raffaella, Stanić Zoran
doaj   +1 more source

Circular RNA expression landscapes in myelodysplastic neoplasms: Associations with mutational signatures and disease progression

open access: yesMolecular Oncology, EarlyView.
In this explorative study, the abundance of circular RNA molecules in bone marrow stem cells was found to be elevated in patients with high‐risk myelodysplastic neoplasms, and to be associated with an increased risk of progression to acute myeloid leukemia.
Eileen Wedge   +17 more
wiley   +1 more source

EDNRB‐dependent endothelin signaling reduces proliferation and promotes proneural‐to‐mesenchymal transition in gliomas

open access: yesMolecular Oncology, EarlyView.
Glioma cells mainly express the endothelin receptor EDNRB, while EDNRA is restricted to a perivascular tumor subpopulation. Endothelin signaling reduces glioma cell proliferation while promoting migration and a proneural‐to‐mesenchymal transition associated with poor prognosis. This pathway activates Ca2+, K+, ERK, and STAT3 signalings and is regulated
Donovan Pineau   +36 more
wiley   +1 more source

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