Results 61 to 70 of about 412,412 (307)

Design methods in the application of fractal geometry in design of women printed fabrics

open access: yesJournal of Architecture, Art & Humanistic Science, 2018
Design is one of the important activities for which the designer studies many different sciences, artistic and philosophical theories, and the most important stages of design.
Shaimaa Shaker
doaj   +1 more source

Geometry of self-similar measures [PDF]

open access: yes, 1995
Las medidas autosemejantes pueden obtenerse considerando el conjunto autosemejante generado por un sistema de semejanzas 1J.i = {
Manuel Morán Cabré   +1 more
openaire   +4 more sources

Cell wall target fragment discovery using a low‐cost, minimal fragment library

open access: yesFEBS Letters, EarlyView.
LoCoFrag100 is a fragment library made up of 100 different compounds. Similarity between the fragments is minimized and 10 different fragments are mixed into a single cocktail, which is soaked to protein crystals. These crystals are analysed by X‐ray crystallography, revealing the binding modes of the bound fragment ligands.
Kaizhou Yan   +5 more
wiley   +1 more source

Magnetic collimation of meridional-self-similar general relativistic MHD flows [PDF]

open access: yes, 2014
We present a model for the spine of relativistic MHD outflows in the Kerr geometry. Meridional self-similarity is invoked to derive semi-analytical solutions close to the polar axis.
Cayatte, Véronique   +3 more
core   +1 more source

Cell density–dependent nuclear‐cytoplasmic shuttling of SETDB1 integrates with Hippo signaling to regulate YAP1‐mediated transcription

open access: yesFEBS Letters, EarlyView.
At low cell density, SETDB1 and YAP1 accumulate in the nucleus. As cell density increases, the Hippo pathway is gradually activated, and SETDB1 is associated with increased YAP1 phosphorylation. At high cell density, phosphorylated YAP1 is sequestered in the cytoplasm, while SETDB1 becomes polyubiquitinated and degraded by the ubiquitin–proteasome ...
Jaemin Eom   +3 more
wiley   +1 more source

Fractals, coherent states and self-similarity induced noncommutative geometry

open access: yes, 2012
The self-similarity properties of fractals are studied in the framework of the theory of entire analytical functions and the $q$-deformed algebra of coherent states. Self-similar structures are related to dissipation and to noncommutative geometry in the
Vitiello, Giuseppe
core   +1 more source

Dual targeting of RET and SRC synergizes in RET fusion‐positive cancer cells

open access: yesMolecular Oncology, EarlyView.
Despite the strong activity of selective RET tyrosine kinase inhibitors (TKIs), resistance of RET fusion‐positive (RET+) lung cancer and thyroid cancer frequently occurs and is mainly driven by RET‐independent bypass mechanisms. Son et al. show that SRC TKIs significantly inhibit PAK and AKT survival signaling and enhance the efficacy of RET TKIs in ...
Juhyeon Son   +13 more
wiley   +1 more source

Review article: methods of fractal geometry used in the study of complex geomorphic networks

open access: yesActa Universitatis Carolinae Geographica, 2014
Fractal geometry methods allow one to quantitatively describe self-similar or self-affined landscape shapes and facilitate the complex/holistic study of natural objects in various scales.
Michal Kusák
doaj   +1 more source

LDAcoop: Integrating non‐linear population dynamics into the analysis of clonogenic growth in vitro

open access: yesMolecular Oncology, EarlyView.
Limiting dilution assays (LDAs) quantify clonogenic growth by seeding serial dilutions of cells and scoring wells for colony formation. The fraction of negative wells is plotted against cells seeded and analyzed using the non‐linear modeling of LDAcoop.
Nikko Brix   +13 more
wiley   +1 more source

Recurrent cancer‐associated ERBB4 mutations are transforming and confer resistance to targeted therapies

open access: yesMolecular Oncology, EarlyView.
We show that the majority of the 18 analyzed recurrent cancer‐associated ERBB4 mutations are transforming. The most potent mutations are activating, co‐operate with other ERBB receptors, and are sensitive to pan‐ERBB inhibitors. Activating ERBB4 mutations also promote therapy resistance in EGFR‐mutant lung cancer.
Veera K. Ojala   +15 more
wiley   +1 more source

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