Results 21 to 30 of about 18,477 (242)

Sinus node dysfunction caused by a sinus node artery occlusion during catheter ablation and sinus rhythm restoration post-PCI [PDF]

open access: yesHeartRhythm Case Reports
Kenji Yamaji, MD, PhD   +5 more
doaj   +2 more sources

Sinus arrest in a p.Arg160X-DSP-positive patient without evidence of desmoplakin-mediated cardiomyopathy: a case report

open access: yesFrontiers in Cardiovascular Medicine, 2023
BackgroundPathogenic/Likely pathogenic variants in DSP-encoded desmoplakin are strongly associated with arrhythmogenic cardiomyopathy (ACM). However, their contribution towards sinus node dysfunction has not been well-delineated.Case summaryA 74-year-old
Nicholas Y. Tan   +4 more
doaj   +1 more source

Sick Sinus Syndrome and Takotsubo Cardiomyopathy

open access: yesCase Reports in Cardiology, 2018
Background. Takotsubo cardiomyopathy is associated with increased risk of ventricular arrhythmias, atrial fibrillation, and bradyarrhythmias. However, sinus node dysfunction is relatively infrequent in the setting of takotsubo cardiomyopathy. Case Report.
Ahmed S. Yassin   +4 more
doaj   +1 more source

Lacosamide-induced sinus node dysfunction followed by severe agranulocytosis

open access: yesBMC Neurology, 2021
Background Lacosamide (LCM) is the antiepileptic drug approved by the U.S. Food and Drug Administration in 2008 that facilitates slow activation of the voltage-gated sodium channels.
Makoto Shibata   +5 more
doaj   +1 more source

Electrocution induced symptomatic bradycardia necessitating pacemaker implantation

open access: yesHeart Views, 2014
Electrical or electrocution injury is a common accidental occurrence and mostly workplace related. Fatal arrhythmias, skin injury and sudden death may ensue.
Kuan Leong Yew
doaj   +1 more source

Brugada syndrome presenting as atrial flutter with sick sinus syndrome

open access: yesHeart India, 2021
An incidentally diagnosed patient with Brugada syndrome was found to have atrial flutter on baseline electrocardiogram. He was DC (Direct current) cardioverted which revealed sick sinus syndrome.
Rohit Rai   +2 more
doaj   +1 more source

Use of machine learning and Poincaré density grid in the diagnosis of sinus node dysfunction caused by sinoatrial conduction block in dogs

open access: yesJournal of Veterinary Internal Medicine
Background Sinus node dysfunction because of abnormal impulse generation or sinoatrial conduction block causes bradycardia that can be difficult to differentiate from high parasympathetic/low sympathetic modulation (HP/LSM).
Wyatt Hutson Flanders   +2 more
doaj   +1 more source

Bioengineered Exosome‐Loaded Immunomodulatory Bioadhesive Spray Reverses Liver Fibrosis and Metabolic Dysfunction That Triggers Beneficial Gut‐Liver Crosstalk in Chronic Fatty Liver Disease

open access: yesAdvanced Science, EarlyView.
Umbilical cord stem cell‐derived exosomes encapsulated in an immunomodulatory bioadhesive hydrogel spray enable localized therapy, sustained vesicle delivery, macrophage polarization, suppress systemic inflammation, and mitigate glucose and insulin homeostasis impairments in MAFLD.
Triya Saha   +4 more
wiley   +1 more source

The Jailed Sinoatrial Node: An Interesting Case of Cardiogenic Shock Secondary to Sinus Arrest Following Percutaneous Intervention

open access: yesJournal of Investigative Medicine High Impact Case Reports, 2022
Complete occlusion of the sinoatrial node artery can be a complication of percutaneous intervention (PCI) to the right coronary artery (RCA). When this happens, dysfunction of the sinus node may follow resulting in sinus arrest.
Ebubechukwu Ezeh MD   +6 more
doaj   +1 more source

A Blood‐Derived Factor Rescues ALS: Platelet Factor 4 Activates OPTN‐Dependent Autophagy to Clear SOD1 Aggregates Independently of PINK1

open access: yesAdvanced Science, EarlyView.
Systemic platelet factor 4 (PF4) is significantly depleted in amyotrophic lateral sclerosis (ALS). Peripheral PF4 replenishment restores central proteostasis by driving OPTN‐dependent, PINK1‐independent selective autophagy in motor neurons. This intervention effectively clears toxic SOD1 aggregates, blunts glial activation, and preserves neuromuscular ...
Qingjian Xie   +12 more
wiley   +1 more source

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