Results 301 to 310 of about 164,723 (328)
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Three Fatal Sodium Azide Poisonings
Medical Toxicology and Adverse Drug Experience, 1989We report 3 cases and review the published literature on sodium azide ingestion. A 38-year-old man intentionally ingested 2 tablespoonsful of sodium azide in water and developed seizures, coma, hypotension and fatal ventricular arrhythmias within 2 hours. A 33-year-old male ingested an unknown quantity of sodium azide.
Brian P. Massaro +5 more
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Thermal analysis of sodium azide
Journal of Hazardous Materials, 1994Abstract The thermal reaction and decomposition of sodium azide were affected by the gas species, its pressure, sample type (powdered or tablet), par- ticle size, sample weight, surface heterogeneity, additives, aging, etc. In actual experiments, very complicated problems arose because those effects were combined together.
Kohzi Ochi +3 more
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Journal of Physics and Chemistry of Solids, 1963
Abstract : This report includes: Measurement of stacking fault probabilities in bulk specimens by Henry M. Otte, D. O. Welch and G. F. Bolling. In x-ray diffraction measurements of NaN3 it has been noted that after deformation, the line broadening was consistent with that expected from formation of deformation stacking faults.
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Abstract : This report includes: Measurement of stacking fault probabilities in bulk specimens by Henry M. Otte, D. O. Welch and G. F. Bolling. In x-ray diffraction measurements of NaN3 it has been noted that after deformation, the line broadening was consistent with that expected from formation of deformation stacking faults.
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Sodium Azide - The Federal Responsibility
SAE Technical Paper Series, 1979The authors discuss present and future Federal involvement in decisions relating to the use of sodium azide to generate gas for air bags and stress the need to establish a perspective from which to examine the raw data concerning the associated health hazards. Several examples are given of other chemicals which present problems in the manufacture, use,
William R. S. Fan, Bruce C. Buckheit
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Fatal Self-Administration of Sodium Azide
Annals of Internal Medicine, 1975A 19-year old woman ingested an unknown amount of sodium azide (NaN3). The earliest symptoms were nausea and loss of vision. Within a few hours her clinical features were dominated by central nervous system signs, acute pulmonary edema, lactic acidosis, and hypothermia.
Julie A. Ricking, Edward A. Emmett
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A Case of Fatal Sodium Azide Ingestion
Journal of Toxicology: Clinical Toxicology, 1986A fatal case of sodium azide poisoning in which exchange blood transfusions, charcoal hemoperfusion, hemodialysis and potent vasopressor agents failed to prevent the development of circulatory collapse associated with a wide complex cardiac rhythm is presented.
Stephanie Reed +2 more
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A case of fatal intoxication with sodium azide
Fühner-Wielands Sammlung von Vergiftungsfällen Archiv für Toxikologie, 1966A case of fatal intoxication with sodium azide is described. The poison was consumed in form of a soluble in water powder. The cause of decease was not explained by the section of the corpse nor by the supplementary microscopic examination. Chemical analysis demonstrated the presence of sodium azide in the stomach contend and small intestine.
J Brzyski, H Koźlicka-Gajdzińska
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Identification of Colloidal Sodium in Sodium Azide
The Journal of Chemical Physics, 1960Under treatment with ultraviolet radiation and heat, sodium azide develops an optical absorption band centered at 520 mμ. The centers responsible for this band have been identified by the dual techniques of electron spin resonance and cryoabsorption spectroscopy as colloidal sodium.
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Sodium Azide-Induced Neurotoxicity
2000Neurodegeneration can be caused by dysfunction of the mitochondrial electron transport chain (ETC), and thus a mitochondrial etiology has been suggested for many neurodegenerative disorders, such as Parkinson’s disease (PD), Huntington’s disease (HD), and Alzheimer’s disease (AD) (1,2).
Yun Wang, Cesario V. Borlongan
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Effects of Sodium Azide on Platelet Function
Thrombosis and Haemostasis, 1977SummarySodium azide in low concentrations (0.1-10 μM) was found to have inhibitory effects on human platelet function. Primary aggregation induced by ADP, epinephrine, thrombin and the ionophore A 23187 was decreased. To evaluate the effect of azide apart from secondary processes, the platelets were treated with indomethacin to prevent prostaglandin ...
Holm Holmsen, Jeanne Stibbe
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