Results 281 to 290 of about 41,599 (347)

Uncovering human kinase substrates in nipah proteome. [PDF]

open access: yesFront Bioinform
Shaji V   +11 more
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Electroencephalographic analysis in soman‐exposed 21‐day‐old rats and the effects of midazolam or LY293558 with caramiphen

Annals of the New York Academy of Sciences, 2020
Acute nerve agent exposure induces status epilepticus (SE), which can cause brain damage or death. Research aiming at developing effective therapies for controlling nerve agent–induced SE is commonly performed in adult rats.
Marcio de Araujo Furtado   +4 more
semanticscholar   +1 more source

Monoclonal antibodies against soman: characterization of soman stereoisomers

Immunology Letters, 1992
Hybridomas were produced which expressed monoclonal anti-soman antibodies as determined by microtiter enzyme-linked-antibody immunoassay (EIA). Each of these antibodies was titrated using a competitive inhibition enzyme immunoassay (CIEIA) with a variety of test ligands. The ligands used included soman (a racemic mixture), sarin, tabun, and each of the
D E, Lenz   +3 more
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Characterization of soman-binding antibodies raised against soman analogs

Molecular Immunology, 1992
The production of antibodies against the highly toxic organophosphorus compound soman (GD) has been undertaken. Monoclonal antibodies were raised against two structural analogs of soman which served as haptens for immunization. In these soman analogs the chemically active P-F bond of the soman molecule was substituted by a P-OH group (which is ionized ...
M, Glikson   +5 more
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Treatment of Poisoning by Soman

Toxicological Sciences, 1985
The efficacies of a number of drug treatment combinations in protecting guinea pigs against the lethal and incapacitating effects of soman (and sarin) have been determined. Incapacitation was studied using a swimming test which is a measure of gross motor performance.
L, Leadbeater, R H, Inns, J M, Rylands
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Naked DNA prevents soman intoxication

Biochemical and Biophysical Research Communications, 2005
Paraoxonase (Q isoenzyme, PON1) can effectively hydrolyze chlorpyrifos-oxon (CPO), soman, sarin, and other organophosphates. Previous studies had indicated that the levels of serum PON1 in gene therapy with adenoviral vector could decrease the toxicity of CPO.
Ai Ling, Fu, Yu Xia, Wang, Man Ji, Sun
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