Tonsil‑derived mesenchymal stem cell‑derived extracellular vesicles suppress MAPK‑NF‑κB signaling and restore osteogenic differentiation in LPS‑stimulated periodontal ligament fibroblasts. [PDF]
Bae WJ, Kim SK, Kim HS, Kang SW, Ban JY.
europepmc +1 more source
The role of bone-derived factors in bone and muscle communication. [PDF]
Li G, Qi M, Liang S.
europepmc +1 more source
Inhibition of sclerostin activity promotes bone regeneration in experimental periodontal disease: A systematic review and meta-analysis of animal models. [PDF]
Yang M, Ma Y, Qu X, Qu Z, Sun J.
europepmc +1 more source
Causal genes for osteoporosis: Mendelian randomization analysis with multilayer xQTL data. [PDF]
Yu F, Wan X, Qiu J.
europepmc +1 more source
Early Diagnosis and Follow-Up of a Novel Homozygous Mutation in <i>SOST</i> Gene in a Child with Recurrent Facial Palsy: A Case Report and Review of the Literature. [PDF]
Acquaviva F +11 more
europepmc +1 more source
Postmenopausal osteoporosis and vascular calcification: The estrogen regulation network and calcification paradox. [PDF]
Lin Y, Jiang P, Lv Y, Tian W.
europepmc +1 more source
Wnt/β-Catenin Pathway and Hydraulic Calcium Silicate-Based Cements: A Narrative Review. [PDF]
Del Giudice C +6 more
europepmc +1 more source
Bone Mineral Density and Serum Levels of Bone Remodeling Markers in Ankylosing Spondylitis Treated with Anti TNF-α Agents. [PDF]
Alvarez-Ayala EG +15 more
europepmc +1 more source
Genetic evidence that SOST inhibits WNT signaling in the limb [PDF]
SOST is a negative regulator of bone formation, and mutations in human SOST are responsible for sclerosteosis. In addition to high bone mass, sclerosteosis patients occasionally display hand defects, suggesting that SOST may function embryonically. Here we report that overexpression of SOST leads to loss of posterior structures of the zeugopod and ...
Gabriela Loots +2 more
exaly +4 more sources

