Results 171 to 180 of about 14,731 (217)
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A Boy With Spastic Paraparesis and Dyspnea

Journal of Child Neurology, 2004
A 4½-year-old boy with signs and symptoms of spastic paraparesis and dyspnea is presented. Biotinidase deficiency was considered and was confirmed by both urine organic acid analysis and biotinidase activity measurement. The child recovered gradually on biotin therapy.
H Serap, Kalkanoğlu   +5 more
openaire   +2 more sources

Progressive Spastic Paraparesis

Archives of Neurology, 1977
To the Editor.— Relative to the article "Progressive Spastic Paraparesis and Adrenal Insufficiency" by Gumbinas and associates published in the October issue of theArchives(33:678, 1976), I take the liberty to add that in the article "Atrophie idiopathique des surrenales et sclerose combinee de la moelle epiniere" by M. Bedivan, A.
openaire   +2 more sources

Hepatic myelopathy with spastic paraparesis

Clinical Neurology and Neurosurgery, 2005
Progressive myelopathy is a rare neurological complication of chronic liver disease with portal hypertension and there is no special diagnostic tool for hepatic myelopathy. Neuropathological studies of the patients with hepatic myelopathy have demonstrated demyelination of the lateral corticospinal tracts with various degree of axonal loss ...
Utku, U   +4 more
openaire   +3 more sources

Spastic paraparesis and sensory neuropathy

Muscle & Nerve, 1992
A 12-year-old developed a slowly progressive spastic gait at the age of 3. A marked loss of pain and temperature sensations led to a mutilating acropathy starting at age 5. Electrodiagnostic studies revealed a symmetric, axonal, predominantly sensory neuropathy, and magnetic resonance imaging ruled out compression of spinal cord.
S N, Tenembaum   +3 more
openaire   +2 more sources

Tropical spastic paraparesis in colombia

Annals of Neurology, 1988
A high-incidence focus of tropical spastic paraparesis (TSP) occurs on the South Pacific coast of Colombia. Of 55 patients studied, 52 (94.5%) had IgG antibodies to the human T-cell lymphotropic virus type I (HTLV-I) in serum and/or cerebrospinal fluid. Control groups did not show similar high positivity.
Vladimir Zaninovic   +11 more
openaire   +2 more sources

The neuroepidemiology of tropical spastic paraparesis

Annals of Neurology, 1988
Recent neuroepidemiological studies of endemic tropical spastic paraparesis (TSP) have confirmed the existence of high-prevalence foci in several tropical islands, including Jamaica and Martinique in the Caribbean, Tumaco off the Pacific coast of Colombia, and the Seychelles in the Indian Ocean.
openaire   +2 more sources

Spastic paraparesis as a manifestation of Leber’s disease

Journal of Neurology, 2005
Sirs: Leber’s hereditary optic neuropathy (LHON) is a genetic disease due to mutations in mitochondrial DNA (mtDNA) responsible for central, bilateral, painless, and fast progressive visual loss with optic nerve atrophy. Various extra-ocular manifestations, called Leber’s “plus” syndrome, have been described [4].
Frédéric, Clarençon   +7 more
openaire   +2 more sources

Immunological studies in tropical spastic paraparesis

Annals of Neurology, 1990
AbstractTropical spastic paraparesis (TSP) and other chronic‐progressive myelopathies have been clearly associated with increased serum and cerebrospinal fluid antibody titers to human T‐lymphotropic virus type I (HTLV‐I). However, little is known about the cellular immune function in TSP. In the present study, activated T lymphocytes were found in the
S, Jacobson   +4 more
openaire   +2 more sources

The Effects of Dalfampridine on Hereditary Spastic Paraparesis

European Neurology, 2016
[No abstract available]
Uygunoglu, Ugur   +5 more
openaire   +3 more sources

Progressive Spastic Paraparesis and Adrenal Insufficiency

Archives of Neurology, 1976
A 10-year-old boy with progressive paraparesis, personality change, and seizures had laboratory evidence of adrenal insufficiency. Pathologic study showed cerebral edema, but no loss of myelin. Notable pathologic changes were limited to the spinal cord, where the corticospinal and spinocerebellar tracts were demyelinated.
M, Gumbinas   +4 more
openaire   +2 more sources

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