Results 61 to 70 of about 147,177 (327)

Pre- and Post-alpha Motoneuronal Control of the Soleus H-reflex during Sinusoidal Hip Movements in Human Spinal Cord Injury [PDF]

open access: yes, 2006
The aim of this study was to establish the contribution of hip-mediated sensory feedback to spinal interneuronal circuits during dynamic conditions in people with incomplete spinal cord injury (SCI).
Chaudhuri, Debjani   +3 more
core   +2 more sources

Age‐Related Characteristics of SYT1‐Associated Neurodevelopmental Disorder

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objectives We describe the clinical manifestations and developmental abilities of individuals with SYT1‐associated neurodevelopmental disorder (Baker‐Gordon syndrome) from infancy to adulthood. We further describe the neuroradiological and electrophysiological characteristics of the condition at different ages, and explore the associations ...
Sam G. Norwitz   +3 more
wiley   +1 more source

Spasticity

open access: yes
Winkle MJ, Huang D, Lui F.
europepmc   +2 more sources

Non-pharmacological interventions for spasticity in adults: An overview of systematic reviews.

open access: yesAnnals of Physical and Rehabilitation Medicine, 2017
OBJECTIVES Spasticity causes significant long-term disability-burden, requiring comprehensive management. This review evaluates evidence from published systematic reviews of clinical trials for effectiveness of non-pharmacological interventions for ...
F. Khan   +3 more
semanticscholar   +1 more source

Impaired H-Reflex Adaptations Following Slope Walking in Individuals With Post-stroke Hemiparesis [PDF]

open access: yes, 2019
Background and Purpose: Short term adaptations in the Ia afferent-motoneuron pathway, as measured using the H-reflex, in response to altered ground reaction forces (GRFs) applied at the feet during slope walking have been observed in the non-impaired ...
Akoopie, Eric   +5 more
core   +2 more sources

Super‐Refractory Status Epilepticus (SRSE) in a Patient With Compound Heterozygous OPA1 Variants: Case Report and Literature Review

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objective Super‐Refractory Status Epilepticus (SRSE) is a rare, life‐threatening neurological emergency with unclear etiology in many cases. Mitochondrial dysfunction, often due to disease‐causing genetic variants, is increasingly recognized as a cause, with each gene producing distinct pathophysiological mechanisms.
Pouria Mohammadi   +2 more
wiley   +1 more source

Cryoneurolysis of the Femoral Nerve for Focal Spasticity in an Ambulatory Patient

open access: yesArchives of Rehabilitation Research and Clinical Translation
Introduction: Spasticity of the knee extensors is a common presentation among patients with multiple sclerosis. The resulting stiff leg gait can result in increased risk of falls, heightened energy expenditure during gait, lowered gait speed, and ...
Ève Boissonnault, MD   +4 more
doaj   +1 more source

Long-Term Care Resident Awareness and Interest in Spasticity Treatments

open access: yesGeriatrics, 2021
Spasticity is common in long-term care settings (affecting up to one in three residents), yet it remains under-treated despite safe and effective, Food and Drug Administration (FDA)-approved therapies. One barrier to treatment may be lack of awareness of
Mallory L. Hacker   +7 more
doaj   +1 more source

Mutations in VPS13D lead to a new recessive ataxia with spasticity and mitochondrial defects

open access: yesAnnals of Neurology, 2018
To identify novel causes of recessive ataxias, including spinocerebellar ataxia with saccadic intrusions, spastic ataxias, and spastic paraplegia.
E. Seong   +18 more
semanticscholar   +1 more source

Mutations in SPG11, encoding spatacsin, are a major cause of spastic paraplegia with thin corpus callosum. [PDF]

open access: yes, 2007
Autosomal recessive hereditary spastic paraplegia (ARHSP) with thin corpus callosum (TCC) is a common and clinically distinct form of familial spastic paraplegia that is linked to the SPG11 locus on chromosome 15 in most affected families. We analyzed
AZZEDINE H   +24 more
core  

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